Table of Contents

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M Vranic,
S Morita,
and G Steiner

Insulin resistance in obesity as analyzed by the response of glucose kinetics to glucagon infusion.

Diabetes March 1980 29:169-176; doi:10.2337/diabetes.29.3.169

Abstract

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E J Coetzee,
W P Jackson,
and P A Berman

Ketonuria in pregnancy--with special reference to calorie-restricted food intake in obese diabetics.

Diabetes March 1980 29:177-181; doi:10.2337/diabetes.29.3.177

Abstract

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P Sidenius and
J Jakobsen

Reduced perikaryal volume of lower motor and primary sensory neurons in early experimental diabetes.

Diabetes March 1980 29:182-186; doi:10.2337/diabetes.29.3.182

Abstract

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K Yamashita,
S Yamashita,
H Yasuda,
Y Oka,
and E Ogata

A decreased response of cyclic adenosine monophosphate concentrations to glucagon in liver slices from streptozotocin-induced diabetic rats.

Diabetes March 1980 29:188-192; doi:10.2337/diabetes.29.3.188

Abstract

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V Leclercq-Meyer,
A Herchuelz,
I Valverde,
E Couturier,
J Marchand,
and W J Malaisse

Mode of action of clonidine upon islet function: dissociated effects upon the time course and magnitude of insulin release.

Diabetes March 1980 29:193-200; doi:10.2337/diabetes.29.3.193

Abstract

M Roudier,
B Portha,
and L Picon

Glucocorticoid-induced recovery from streptozotocin diabetes in the adult rat.

Diabetes March 1980 29:201-205

M C Champion,
G A Shepherd,
N W Rodger,
and J Dupre

Continuous subcutaneous infusion of insulin in the management of diabetes mellitus.

Diabetes March 1980 29:206-212

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L J Fischer and
S A Hamburger

Inhibition of alloxan action in isolated pancreatic islets by superoxide dismutase, catalase, and a metal chelator.

Diabetes March 1980 29:213-216; doi:10.2337/diabetes.29.3.213

Abstract

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D E Goldstein and
R T Curnow

Impaired glycogen synthase activating system in human diabetic polymorphonuclear leukocytes.

Diabetes March 1980 29:217-220; doi:10.2337/diabetes.29.3.217

Abstract

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B Draznin,
C C Solomons,
C A Emler,
D S Schalch,
and K E Sussman

Decreased insulin binding and degradation associated with depressed intracellular ATP content.

Diabetes March 1980 29:221-226; doi:10.2337/diabetes.29.3.221

Abstract

R S Clements, Jr and
C R Stockard

Abnormal sciatic nerve myo-inositol metabolism in the streptozotocin-diabetic rat: effect of insulin treatment.

Diabetes March 1980 29:227-235

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J D McGarry and
D W Foster

Effects of exogenous fatty acid concentration on glucagon-induced changes in hepatic fatty acid metabolism.

Diabetes March 1980 29:236-240; doi:10.2337/diabetes.29.3.236

Abstract

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A M Albisser,
W Lougheed,
K Perlman,
and A Bahoric

Nonaggregating insulin solutions for long-term glucose control in experimental and human diabetes.

Diabetes March 1980 29:241-243; doi:10.2337/diabetes.29.3.241

Abstract

Diabetes

Insulin resistance in obesity as analyzed by the response of glucose kinetics to glucagon infusion.

Ketonuria in pregnancy--with special reference to calorie-restricted food intake in obese diabetics.

Reduced perikaryal volume of lower motor and primary sensory neurons in early experimental diabetes.

A decreased response of cyclic adenosine monophosphate concentrations to glucagon in liver slices from streptozotocin-induced diabetic rats.

Mode of action of clonidine upon islet function: dissociated effects upon the time course and magnitude of insulin release.

Glucocorticoid-induced recovery from streptozotocin diabetes in the adult rat.

Continuous subcutaneous infusion of insulin in the management of diabetes mellitus.

Inhibition of alloxan action in isolated pancreatic islets by superoxide dismutase, catalase, and a metal chelator.

Impaired glycogen synthase activating system in human diabetic polymorphonuclear leukocytes.

Decreased insulin binding and degradation associated with depressed intracellular ATP content.

Abnormal sciatic nerve myo-inositol metabolism in the streptozotocin-diabetic rat: effect of insulin treatment.

Effects of exogenous fatty acid concentration on glucagon-induced changes in hepatic fatty acid metabolism.

Nonaggregating insulin solutions for long-term glucose control in experimental and human diabetes.

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