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Metabolic consequences of sustained suppression of free fatty acids by acipimox in patients with NIDDM.

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Postprandial free fatty acid kinetics are abnormal in upper body obesity.

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Complement lytic activity has no role in the pathogenesis of autoimmune diabetes in NOD mice.

• R P Ferraris,
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Dietary carbohydrate enhances intestinal sugar transport in diabetic mice.

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Effects of insulin on fatty acid reesterification in healthy subjects.

• F Sakakibara,
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Effects of high glucose concentrations and epalrestat on sorbitol and myo-inositol metabolism in cultured rabbit aortic smooth muscle cells.

• N M Parish,
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Tolerance to IDDM induced by CD4 antibodies in nonobese diabetic mice is reversed by cyclophosphamide.

• N Di Girolamo,
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Functional activity of plasma fibronectin in patients with diabetes mellitus.

• L B Gardner,
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The role of glucose-6-phosphatase in the action of insulin on hepatic glucose production in the rat.

• B T Jackson,
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Alcohol intake impairs glucose counterregulation during acute insulin-induced hypoglycemia in IDDM patients. Evidence for a critical role of free fatty acids.

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Reduced sampling protocols in estimation of insulin sensitivity and glucose effectiveness using the minimal model in NIDDM.

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Cytoplasmic islet cell antibodies recognize distinct islet antigens in IDDM but not in stiff man syndrome.

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The pharmacodynamics and activity of intranasally administered insulin in healthy male volunteers.

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Endothelial cell-binding properties of lymphocytes infiltrated into human diabetic pancreas. Implications for pathogenesis of IDDM.

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Quantification of the relationship between insulin sensitivity and beta-cell function in human subjects. Evidence for a hyperbolic function.

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Transforming growth factor-beta 1 enhances glomerular collagen synthesis in diabetic rats.

• B Thorens,
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Cloning and functional expression of the human islet GLP-1 receptor. Demonstration that exendin-4 is an agonist and exendin-(9-39) an antagonist of the receptor.

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Meticulous prevention of hypoglycemia normalizes the glycemic thresholds and magnitude of most of neuroendocrine responses to, symptoms of, and cognitive function during hypoglycemia in intensively treated patients with short-term IDDM.