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Index by Author

January 1994; 43 (1)
  1. A
  2. B
  3. C
  4. D
  5. E
  6. F
  7. G
  8. H
  9. I
  10. J
  11. K
  12. L
  13. M
  14. N
  15. O
  16. P
  17. Q
  18. R
  19. S
  20. T
  21. U
  22. V
  23. W
  24. X
  25. Y
  26. Z

A

  1. Akazawa, S

    • Autoantibodies to glutamic acid decarboxylase in patients with IDDM and autoimmune thyroid disease. Diabetes January 1994 43:80-86; doi:10.2337/diabetes.43.1.80

  2. Arfken, C L

    • Factors responsible for impaired fibrinolysis in obese subjects and NIDDM patients. Diabetes January 1994 43:104-109; doi:10.2337/diabetes.43.1.104

B

  1. Bailey, R C

    • Growth hormone induces resistance to the mitogenic action of insulin through local IGF-I. Studies in normal and Pygmy T-cell lines. Diabetes January 1994 43:68-72; doi:10.2337/diabetes.43.1.68

  2. Balkau, B

    • Maternal effect and familial aggregation in NIDDM. The CODIAB Study. CODIAB-INSERM-ZENECA Study Group. Diabetes January 1994 43:63-67; doi:10.2337/diabetes.43.1.63

  3. Bao, S

    • Biological actions of insulin are differentially regulated by glucose and insulin in primary cultured adipocytes. Chronic ability to increase glycogen synthase activity. Diabetes January 1994 43:53-62; doi:10.2337/diabetes.43.1.53

  4. Bell, G I

    • Abnormal insulin secretion, not insulin resistance, is the genetic or primary defect of MODY in the RW pedigree. Diabetes January 1994 43:40-46; doi:10.2337/diabetes.43.1.40

  5. Bellgrau, D

    • Peculiar T-cell signaling does not preclude positive selection in the diabetes-prone BB rat. Diabetes January 1994 43:47-52; doi:10.2337/diabetes.43.1.47

  6. Bersch, N

    • Growth hormone induces resistance to the mitogenic action of insulin through local IGF-I. Studies in normal and Pygmy T-cell lines. Diabetes January 1994 43:68-72; doi:10.2337/diabetes.43.1.68

  7. Bertrand, S

    • Predictive value of lymphocyte antibodies for the appearance of diabetes in BB rats. Diabetes January 1994 43:137-142; doi:10.2337/diabetes.43.1.137

  8. Björk, E

    • GAD autoantibodies in IDDM, stiff-man syndrome, and autoimmune polyendocrine syndrome type I recognize different epitopes. Diabetes January 1994 43:161-165; doi:10.2337/diabetes.43.1.161

  9. Buelow, R

    • Isolation of nonobese diabetic mouse T-cells that recognize novel autoantigens involved in the early events of diabetes. Diabetes January 1994 43:33-39; doi:10.2337/diabetes.43.1.33

C

  1. Christie, M R

    • Associations of anti-GAD antibodies with islet cell antibodies and insulin autoantibodies in first-degree relatives of type I diabetic patients. Diabetes January 1994 43:154-160; doi:10.2337/diabetes.43.1.154

  2. Cox, N J

    • Maternal component in NIDDM transmission. How large an effect? Diabetes January 1994 43:166-168

  3. Craven, P A

    • Activation of protein kinase C in glomerular cells in diabetes. Mechanisms and potential links to the pathogenesis of diabetic glomerulopathy. Diabetes January 1994 43:1-8; doi:10.2337/diabetes.43.1.1

D

  1. Derubertis, F R

    • Activation of protein kinase C in glomerular cells in diabetes. Mechanisms and potential links to the pathogenesis of diabetic glomerulopathy. Diabetes January 1994 43:1-8; doi:10.2337/diabetes.43.1.1

  2. Dryden, S

    • Effects of chronic vanadate administration in the STZ-induced diabetic rat. The antihyperglycemic action of vanadate is attributable entirely to its suppression of feeding. Diabetes January 1994 43:9-15; doi:10.2337/diabetes.43.1.9

E

  1. Ellis, L

    • A new transgenic mouse model of chronic hyperglycemia. Diabetes January 1994 43:143-153; doi:10.2337/diabetes.43.1.143

F

  1. Fajans, S S

    • Abnormal insulin secretion, not insulin resistance, is the genetic or primary defect of MODY in the RW pedigree. Diabetes January 1994 43:40-46; doi:10.2337/diabetes.43.1.40

  2. Fathman, C G

    • Isolation of nonobese diabetic mouse T-cells that recognize novel autoantigens involved in the early events of diabetes. Diabetes January 1994 43:33-39; doi:10.2337/diabetes.43.1.33

  3. Ferré, P

    • A new transgenic mouse model of chronic hyperglycemia. Diabetes January 1994 43:143-153; doi:10.2337/diabetes.43.1.143

  4. Finke, E H

    • Induction of tolerance to islet xenografts in a concordant rat-to-mouse model. Diabetes January 1994 43:16-23; doi:10.2337/diabetes.43.1.16

  5. Flye, M W

    • Induction of tolerance to islet xenografts in a concordant rat-to-mouse model. Diabetes January 1994 43:16-23; doi:10.2337/diabetes.43.1.16

  6. Foot, E A

    • Effects of calcium antagonists on insulin-mediated glucose metabolism in skeletal muscle. Diabetes January 1994 43:73-79; doi:10.2337/diabetes.43.1.73

  7. Freidenberg, G R

    • Delayed onset of insulin activation of the insulin receptor kinase in vivo in human skeletal muscle. Diabetes January 1994 43:118-126; doi:10.2337/diabetes.43.1.118

  8. Fugger, L

    • Major histocompatibility complex-encoded antigen processing gene polymorphism in IDDM. Diabetes January 1994 43:110-117; doi:10.2337/diabetes.43.1.110

G

  1. Garvey, W T

    • Biological actions of insulin are differentially regulated by glucose and insulin in primary cultured adipocytes. Chronic ability to increase glycogen synthase activity. Diabetes January 1994 43:53-62; doi:10.2337/diabetes.43.1.53

  2. Geffner, M E

    • Growth hormone induces resistance to the mitogenic action of insulin through local IGF-I. Studies in normal and Pygmy T-cell lines. Diabetes January 1994 43:68-72; doi:10.2337/diabetes.43.1.68

  3. Gelber, C

    • Isolation of nonobese diabetic mouse T-cells that recognize novel autoantigens involved in the early events of diabetes. Diabetes January 1994 43:33-39; doi:10.2337/diabetes.43.1.33

  4. Gillison, S L

    • ADP ribosylation by cholera toxin identifies three G-proteins that are activated by the galanin receptor. Studies with RINm5F cell membranes. Diabetes January 1994 43:24-32; doi:10.2337/diabetes.43.1.24

  5. Golde, D W

    • Growth hormone induces resistance to the mitogenic action of insulin through local IGF-I. Studies in normal and Pygmy T-cell lines. Diabetes January 1994 43:68-72; doi:10.2337/diabetes.43.1.68

  6. Goss, J A

    • Induction of tolerance to islet xenografts in a concordant rat-to-mouse model. Diabetes January 1994 43:16-23; doi:10.2337/diabetes.43.1.16

H

  1. Halter, J B

    • Abnormal insulin secretion, not insulin resistance, is the genetic or primary defect of MODY in the RW pedigree. Diabetes January 1994 43:40-46; doi:10.2337/diabetes.43.1.40

  2. Hammer, R E

    • A new transgenic mouse model of chronic hyperglycemia. Diabetes January 1994 43:143-153; doi:10.2337/diabetes.43.1.143

  3. Henry, R R

    • Delayed onset of insulin activation of the insulin receptor kinase in vivo in human skeletal muscle. Diabetes January 1994 43:118-126; doi:10.2337/diabetes.43.1.118

  4. Herman, W H

    • Abnormal insulin secretion, not insulin resistance, is the genetic or primary defect of MODY in the RW pedigree. Diabetes January 1994 43:40-46; doi:10.2337/diabetes.43.1.40

J

  1. Jolicoeur, C

    • Isolation of nonobese diabetic mouse T-cells that recognize novel autoantigens involved in the early events of diabetes. Diabetes January 1994 43:33-39; doi:10.2337/diabetes.43.1.33

K

  1. Kämpe, O

    • GAD autoantibodies in IDDM, stiff-man syndrome, and autoimmune polyendocrine syndrome type I recognize different epitopes. Diabetes January 1994 43:161-165; doi:10.2337/diabetes.43.1.161

  2. Karlsson, F A

    • GAD autoantibodies in IDDM, stiff-man syndrome, and autoimmune polyendocrine syndrome type I recognize different epitopes. Diabetes January 1994 43:161-165; doi:10.2337/diabetes.43.1.161

  3. Kawasaki, E

    • Autoantibodies to glutamic acid decarboxylase in patients with IDDM and autoimmune thyroid disease. Diabetes January 1994 43:80-86; doi:10.2337/diabetes.43.1.80

  4. Kilpatrick, A

    • Effects of chronic vanadate administration in the STZ-induced diabetic rat. The antihyperglycemic action of vanadate is attributable entirely to its suppression of feeding. Diabetes January 1994 43:9-15; doi:10.2337/diabetes.43.1.9

L

  1. Lacy, P E

    • Induction of tolerance to islet xenografts in a concordant rat-to-mouse model. Diabetes January 1994 43:16-23; doi:10.2337/diabetes.43.1.16

  2. Leech, N J

    • High genetic risk for IDDM in the Pacific Northwest. First report from the Washington State Diabetes Prediction Study. Diabetes January 1994 43:87-94; doi:10.2337/diabetes.43.1.87

  3. Leighton, B

    • Effects of calcium antagonists on insulin-mediated glucose metabolism in skeletal muscle. Diabetes January 1994 43:73-79; doi:10.2337/diabetes.43.1.73

  4. Liblau, R S

    • Major histocompatibility complex-encoded antigen processing gene polymorphism in IDDM. Diabetes January 1994 43:110-117; doi:10.2337/diabetes.43.1.110

  5. Lima, F B

    • Biological actions of insulin are differentially regulated by glucose and insulin in primary cultured adipocytes. Chronic ability to increase glycogen synthase activity. Diabetes January 1994 43:53-62; doi:10.2337/diabetes.43.1.53

  6. Lopez, T

    • Major histocompatibility complex-encoded antigen processing gene polymorphism in IDDM. Diabetes January 1994 43:110-117; doi:10.2337/diabetes.43.1.110

  7. Lucore, C L

    • Factors responsible for impaired fibrinolysis in obese subjects and NIDDM patients. Diabetes January 1994 43:104-109; doi:10.2337/diabetes.43.1.104

M

  1. Maika, S D

    • A new transgenic mouse model of chronic hyperglycemia. Diabetes January 1994 43:143-153; doi:10.2337/diabetes.43.1.143

  2. Malabu, U H

    • Effects of chronic vanadate administration in the STZ-induced diabetic rat. The antihyperglycemic action of vanadate is attributable entirely to its suppression of feeding. Diabetes January 1994 43:9-15; doi:10.2337/diabetes.43.1.9

  3. Matsumoto, K

    • Autoantibodies to glutamic acid decarboxylase in patients with IDDM and autoimmune thyroid disease. Diabetes January 1994 43:80-86; doi:10.2337/diabetes.43.1.80

  4. McAteer, D

    • Isolation of nonobese diabetic mouse T-cells that recognize novel autoantigens involved in the early events of diabetes. Diabetes January 1994 43:33-39; doi:10.2337/diabetes.43.1.33

  5. McCarthy, H D

    • Effects of chronic vanadate administration in the STZ-induced diabetic rat. The antihyperglycemic action of vanadate is attributable entirely to its suppression of feeding. Diabetes January 1994 43:9-15; doi:10.2337/diabetes.43.1.9

  6. McCulloch, D K

    • High genetic risk for IDDM in the Pacific Northwest. First report from the Washington State Diabetes Prediction Study. Diabetes January 1994 43:87-94; doi:10.2337/diabetes.43.1.87

  7. McDevitt, H

    • Isolation of nonobese diabetic mouse T-cells that recognize novel autoantigens involved in the early events of diabetes. Diabetes January 1994 43:33-39; doi:10.2337/diabetes.43.1.33

  8. McDevitt, H O

    • Major histocompatibility complex-encoded antigen processing gene polymorphism in IDDM. Diabetes January 1994 43:110-117; doi:10.2337/diabetes.43.1.110

  9. McGill, J B

    • Factors responsible for impaired fibrinolysis in obese subjects and NIDDM patients. Diabetes January 1994 43:104-109; doi:10.2337/diabetes.43.1.104

  10. Morin, J

    • A new transgenic mouse model of chronic hyperglycemia. Diabetes January 1994 43:143-153; doi:10.2337/diabetes.43.1.143

N

  1. Nagataki, S

    • Autoantibodies to glutamic acid decarboxylase in patients with IDDM and autoimmune thyroid disease. Diabetes January 1994 43:80-86; doi:10.2337/diabetes.43.1.80

  2. Nakafusa, Y

    • Induction of tolerance to islet xenografts in a concordant rat-to-mouse model. Diabetes January 1994 43:16-23; doi:10.2337/diabetes.43.1.16

  3. Nepom, G T

    • High genetic risk for IDDM in the Pacific Northwest. First report from the Washington State Diabetes Prediction Study. Diabetes January 1994 43:87-94; doi:10.2337/diabetes.43.1.87

  4. Nerup, J

    • Major histocompatibility complex-encoded antigen processing gene polymorphism in IDDM. Diabetes January 1994 43:110-117; doi:10.2337/diabetes.43.1.110

  5. Nolan, J

    • Delayed onset of insulin activation of the insulin receptor kinase in vivo in human skeletal muscle. Diabetes January 1994 43:118-126; doi:10.2337/diabetes.43.1.118

O

  1. Olefsky, J M

    • Delayed onset of insulin activation of the insulin receptor kinase in vivo in human skeletal muscle. Diabetes January 1994 43:118-126; doi:10.2337/diabetes.43.1.118

  2. Ortiz, F J

    • Abnormal insulin secretion, not insulin resistance, is the genetic or primary defect of MODY in the RW pedigree. Diabetes January 1994 43:40-46; doi:10.2337/diabetes.43.1.40

P

  1. Paborsky, L

    • Isolation of nonobese diabetic mouse T-cells that recognize novel autoantigens involved in the early events of diabetes. Diabetes January 1994 43:33-39; doi:10.2337/diabetes.43.1.33

  2. Papoz, L

    • Maternal effect and familial aggregation in NIDDM. The CODIAB Study. CODIAB-INSERM-ZENECA Study Group. Diabetes January 1994 43:63-67; doi:10.2337/diabetes.43.1.63

  3. Patel, S D

    • Major histocompatibility complex-encoded antigen processing gene polymorphism in IDDM. Diabetes January 1994 43:110-117; doi:10.2337/diabetes.43.1.110

  4. Pénicaud, L

    • A new transgenic mouse model of chronic hyperglycemia. Diabetes January 1994 43:143-153; doi:10.2337/diabetes.43.1.143

  5. Pilkey, D T

    • Identification in islets of Langerhans of a new rat alpha 2-adrenergic receptor. Diabetes January 1994 43:127-136; doi:10.2337/diabetes.43.1.127

  6. Pociot, F

    • Major histocompatibility complex-encoded antigen processing gene polymorphism in IDDM. Diabetes January 1994 43:110-117; doi:10.2337/diabetes.43.1.110

  7. Polonsky, K S

    • Abnormal insulin secretion, not insulin resistance, is the genetic or primary defect of MODY in the RW pedigree. Diabetes January 1994 43:40-46; doi:10.2337/diabetes.43.1.40

R

  1. Ramos, P

    • A new transgenic mouse model of chronic hyperglycemia. Diabetes January 1994 43:143-153; doi:10.2337/diabetes.43.1.143

  2. Redd, J M

    • Peculiar T-cell signaling does not preclude positive selection in the diabetes-prone BB rat. Diabetes January 1994 43:47-52; doi:10.2337/diabetes.43.1.47

  3. Reichart, D

    • Delayed onset of insulin activation of the insulin receptor kinase in vivo in human skeletal muscle. Diabetes January 1994 43:118-126; doi:10.2337/diabetes.43.1.118

  4. Roll, U

    • Associations of anti-GAD antibodies with islet cell antibodies and insulin autoantibodies in first-degree relatives of type I diabetic patients. Diabetes January 1994 43:154-160; doi:10.2337/diabetes.43.1.154

  5. Rowe, R E

    • High genetic risk for IDDM in the Pacific Northwest. First report from the Washington State Diabetes Prediction Study. Diabetes January 1994 43:87-94; doi:10.2337/diabetes.43.1.87

S

  1. Schaefer, E M

    • A new transgenic mouse model of chronic hyperglycemia. Diabetes January 1994 43:143-153; doi:10.2337/diabetes.43.1.143

  2. Schneider, D J

    • Factors responsible for impaired fibrinolysis in obese subjects and NIDDM patients. Diabetes January 1994 43:104-109; doi:10.2337/diabetes.43.1.104

  3. Sellins, K S

    • Peculiar T-cell signaling does not preclude positive selection in the diabetes-prone BB rat. Diabetes January 1994 43:47-52; doi:10.2337/diabetes.43.1.47

  4. Sharp, G W

    • ADP ribosylation by cholera toxin identifies three G-proteins that are activated by the galanin receptor. Studies with RINm5F cell membranes. Diabetes January 1994 43:24-32; doi:10.2337/diabetes.43.1.24

  5. Singer, S

    • Isolation of nonobese diabetic mouse T-cells that recognize novel autoantigens involved in the early events of diabetes. Diabetes January 1994 43:33-39; doi:10.2337/diabetes.43.1.33

  6. Smith, M J

    • Abnormal insulin secretion, not insulin resistance, is the genetic or primary defect of MODY in the RW pedigree. Diabetes January 1994 43:40-46; doi:10.2337/diabetes.43.1.40

  7. Sobel, B E

    • Factors responsible for impaired fibrinolysis in obese subjects and NIDDM patients. Diabetes January 1994 43:104-109; doi:10.2337/diabetes.43.1.104

  8. Spiro, M J

    • Synthesis of type VI collagen by cultured glomerular cells and comparison of its regulation by glucose and other factors with that of type IV collagen. Diabetes January 1994 43:95-103; doi:10.2337/diabetes.43.1.95

  9. Spiro, R G

    • Synthesis of type VI collagen by cultured glomerular cells and comparison of its regulation by glucose and other factors with that of type IV collagen. Diabetes January 1994 43:95-103; doi:10.2337/diabetes.43.1.95

  10. Standl, E

    • Associations of anti-GAD antibodies with islet cell antibodies and insulin autoantibodies in first-degree relatives of type I diabetic patients. Diabetes January 1994 43:154-160; doi:10.2337/diabetes.43.1.154

  11. Sturis, J

    • Abnormal insulin secretion, not insulin resistance, is the genetic or primary defect of MODY in the RW pedigree. Diabetes January 1994 43:40-46; doi:10.2337/diabetes.43.1.40

  12. Suter, S

    • Delayed onset of insulin activation of the insulin receptor kinase in vivo in human skeletal muscle. Diabetes January 1994 43:118-126; doi:10.2337/diabetes.43.1.118

T

  1. Takao, Y

    • Autoantibodies to glutamic acid decarboxylase in patients with IDDM and autoimmune thyroid disease. Diabetes January 1994 43:80-86; doi:10.2337/diabetes.43.1.80

  2. Takino, H

    • Autoantibodies to glutamic acid decarboxylase in patients with IDDM and autoimmune thyroid disease. Diabetes January 1994 43:80-86; doi:10.2337/diabetes.43.1.80

  3. Thomas, F

    • Maternal effect and familial aggregation in NIDDM. The CODIAB Study. CODIAB-INSERM-ZENECA Study Group. Diabetes January 1994 43:63-67; doi:10.2337/diabetes.43.1.63

  4. Tisch, R

    • Isolation of nonobese diabetic mouse T-cells that recognize novel autoantigens involved in the early events of diabetes. Diabetes January 1994 43:33-39; doi:10.2337/diabetes.43.1.33

U

  1. Uotani, S

    • Autoantibodies to glutamic acid decarboxylase in patients with IDDM and autoimmune thyroid disease. Diabetes January 1994 43:80-86; doi:10.2337/diabetes.43.1.80

V

  1. van Endert, P M

    • Major histocompatibility complex-encoded antigen processing gene polymorphism in IDDM. Diabetes January 1994 43:110-117; doi:10.2337/diabetes.43.1.110

  2. Vauzelle-Kervroedan, F

    • Maternal effect and familial aggregation in NIDDM. The CODIAB Study. CODIAB-INSERM-ZENECA Study Group. Diabetes January 1994 43:63-67; doi:10.2337/diabetes.43.1.63

  3. Velloso, L A

    • GAD autoantibodies in IDDM, stiff-man syndrome, and autoimmune polyendocrine syndrome type I recognize different epitopes. Diabetes January 1994 43:161-165; doi:10.2337/diabetes.43.1.161

  4. Viard, V

    • A new transgenic mouse model of chronic hyperglycemia. Diabetes January 1994 43:143-153; doi:10.2337/diabetes.43.1.143

  5. Vigeant, C

    • Predictive value of lymphocyte antibodies for the appearance of diabetes in BB rats. Diabetes January 1994 43:137-142; doi:10.2337/diabetes.43.1.137

W

  1. Wakisaka, M

    • Synthesis of type VI collagen by cultured glomerular cells and comparison of its regulation by glucose and other factors with that of type IV collagen. Diabetes January 1994 43:95-103; doi:10.2337/diabetes.43.1.95

  2. Wang, S Y

    • Identification in islets of Langerhans of a new rat alpha 2-adrenergic receptor. Diabetes January 1994 43:127-136; doi:10.2337/diabetes.43.1.127

  3. Williams, G

    • Effects of chronic vanadate administration in the STZ-induced diabetic rat. The antihyperglycemic action of vanadate is attributable entirely to its suppression of feeding. Diabetes January 1994 43:9-15; doi:10.2337/diabetes.43.1.9

Y

  1. Yale, J F

    • Predictive value of lymphocyte antibodies for the appearance of diabetes in BB rats. Diabetes January 1994 43:137-142; doi:10.2337/diabetes.43.1.137

  2. Yamaguchi, Y

    • Autoantibodies to glutamic acid decarboxylase in patients with IDDM and autoimmune thyroid disease. Diabetes January 1994 43:80-86; doi:10.2337/diabetes.43.1.80

  3. Yano, M

    • Autoantibodies to glutamic acid decarboxylase in patients with IDDM and autoimmune thyroid disease. Diabetes January 1994 43:80-86; doi:10.2337/diabetes.43.1.80

Z

  1. Ziegler, A G

    • Associations of anti-GAD antibodies with islet cell antibodies and insulin autoantibodies in first-degree relatives of type I diabetic patients. Diabetes January 1994 43:154-160; doi:10.2337/diabetes.43.1.154

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