Table of Contents

Select this article
- F R Derubertis and
- P A Craven
Activation of protein kinase C in glomerular cells in diabetes. Mechanisms and potential links to the pathogenesis of diabetic glomerulopathy.
Diabetes January 1994 43:1-8; doi:10.2337/diabetes.43.1.1
- Abstract
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- U H Malabu,
- S Dryden,
- H D McCarthy,
- A Kilpatrick,
- and G Williams
Effects of chronic vanadate administration in the STZ-induced diabetic rat. The antihyperglycemic action of vanadate is attributable entirely to its suppression of feeding.
Diabetes January 1994 43:9-15; doi:10.2337/diabetes.43.1.9
- Abstract
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- J A Goss,
- Y Nakafusa,
- E H Finke,
- M W Flye,
- and P E Lacy
Induction of tolerance to islet xenografts in a concordant rat-to-mouse model.
Diabetes January 1994 43:16-23; doi:10.2337/diabetes.43.1.16
- Abstract
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- S L Gillison and
- G W Sharp
ADP ribosylation by cholera toxin identifies three G-proteins that are activated by the galanin receptor. Studies with RINm5F cell membranes.
Diabetes January 1994 43:24-32; doi:10.2337/diabetes.43.1.24
- Abstract
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- C Gelber,
- L Paborsky,
- S Singer,
- D McAteer,
- R Tisch,
- C Jolicoeur,
- R Buelow,
- H McDevitt,
- and C G Fathman
Isolation of nonobese diabetic mouse T-cells that recognize novel autoantigens involved in the early events of diabetes.
Diabetes January 1994 43:33-39; doi:10.2337/diabetes.43.1.33
- Abstract
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- W H Herman,
- S S Fajans,
- F J Ortiz,
- M J Smith,
- J Sturis,
- G I Bell,
- K S Polonsky,
- and J B Halter
Abnormal insulin secretion, not insulin resistance, is the genetic or primary defect of MODY in the RW pedigree.
Diabetes January 1994 43:40-46; doi:10.2337/diabetes.43.1.40
- Abstract
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- D Bellgrau,
- J M Redd,
- and K S Sellins
Peculiar T-cell signaling does not preclude positive selection in the diabetes-prone BB rat.
Diabetes January 1994 43:47-52; doi:10.2337/diabetes.43.1.47
- Abstract
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- F B Lima,
- S Bao,
- and W T Garvey
Biological actions of insulin are differentially regulated by glucose and insulin in primary cultured adipocytes. Chronic ability to increase glycogen synthase activity.
Diabetes January 1994 43:53-62; doi:10.2337/diabetes.43.1.53
- Abstract
Select this article
- F Thomas,
- B Balkau,
- F Vauzelle-Kervroedan,
- and L Papoz
Maternal effect and familial aggregation in NIDDM. The CODIAB Study. CODIAB-INSERM-ZENECA Study Group.
Diabetes January 1994 43:63-67; doi:10.2337/diabetes.43.1.63
- Abstract
Select this article
- M E Geffner,
- N Bersch,
- R C Bailey,
- and D W Golde
Growth hormone induces resistance to the mitogenic action of insulin through local IGF-I. Studies in normal and Pygmy T-cell lines.
Diabetes January 1994 43:68-72; doi:10.2337/diabetes.43.1.68
- Abstract
Select this article
- E A Foot and
- B Leighton
Effects of calcium antagonists on insulin-mediated glucose metabolism in skeletal muscle.
Diabetes January 1994 43:73-79; doi:10.2337/diabetes.43.1.73
- Abstract
Select this article
- E Kawasaki,
- H Takino,
- M Yano,
- S Uotani,
- K Matsumoto,
- Y Takao,
- Y Yamaguchi,
- S Akazawa,
- and S Nagataki
Autoantibodies to glutamic acid decarboxylase in patients with IDDM and autoimmune thyroid disease.
Diabetes January 1994 43:80-86; doi:10.2337/diabetes.43.1.80
- Abstract
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- R E Rowe,
- N J Leech,
- G T Nepom,
- and D K McCulloch
High genetic risk for IDDM in the Pacific Northwest. First report from the Washington State Diabetes Prediction Study.
Diabetes January 1994 43:87-94; doi:10.2337/diabetes.43.1.87
- Abstract
Select this article
- M Wakisaka,
- M J Spiro,
- and R G Spiro
Synthesis of type VI collagen by cultured glomerular cells and comparison of its regulation by glucose and other factors with that of type IV collagen.
Diabetes January 1994 43:95-103; doi:10.2337/diabetes.43.1.95
- Abstract
Select this article
- J B McGill,
- D J Schneider,
- C L Arfken,
- C L Lucore,
- and B E Sobel
Factors responsible for impaired fibrinolysis in obese subjects and NIDDM patients.
Diabetes January 1994 43:104-109; doi:10.2337/diabetes.43.1.104
- Abstract
Select this article
- P M van Endert,
- R S Liblau,
- S D Patel,
- L Fugger,
- T Lopez,
- F Pociot,
- J Nerup,
- and H O McDevitt
Major histocompatibility complex-encoded antigen processing gene polymorphism in IDDM.
Diabetes January 1994 43:110-117; doi:10.2337/diabetes.43.1.110
- Abstract
Select this article
- G R Freidenberg,
- S Suter,
- R R Henry,
- J Nolan,
- D Reichart,
- and J M Olefsky
Delayed onset of insulin activation of the insulin receptor kinase in vivo in human skeletal muscle.
Diabetes January 1994 43:118-126; doi:10.2337/diabetes.43.1.118
- Abstract
Select this article
- S Y Wang and
- D T Pilkey
Identification in islets of Langerhans of a new rat alpha 2-adrenergic receptor.
Diabetes January 1994 43:127-136; doi:10.2337/diabetes.43.1.127
- Abstract
Select this article
- S Bertrand,
- C Vigeant,
- and J F Yale
Predictive value of lymphocyte antibodies for the appearance of diabetes in BB rats.
Diabetes January 1994 43:137-142; doi:10.2337/diabetes.43.1.137
- Abstract
Select this article
- E M Schaefer,
- V Viard,
- J Morin,
- P Ferré,
- L Pénicaud,
- P Ramos,
- S D Maika,
- L Ellis,
- and R E Hammer
A new transgenic mouse model of chronic hyperglycemia.
Diabetes January 1994 43:143-153; doi:10.2337/diabetes.43.1.143
- Abstract
Select this article
- U Roll,
- M R Christie,
- E Standl,
- and A G Ziegler
Associations of anti-GAD antibodies with islet cell antibodies and insulin autoantibodies in first-degree relatives of type I diabetic patients.
Diabetes January 1994 43:154-160; doi:10.2337/diabetes.43.1.154
- Abstract
Select this article
- E Björk,
- L A Velloso,
- O Kämpe,
- and F A Karlsson
GAD autoantibodies in IDDM, stiff-man syndrome, and autoimmune polyendocrine syndrome type I recognize different epitopes.
Diabetes January 1994 43:161-165; doi:10.2337/diabetes.43.1.161
- Abstract
- N J Cox
Maternal component in NIDDM transmission. How large an effect?
Diabetes January 1994 43:166-168

Activation of protein kinase C in glomerular cells in diabetes. Mechanisms and potential links to the pathogenesis of diabetic glomerulopathy.

Effects of chronic vanadate administration in the STZ-induced diabetic rat. The antihyperglycemic action of vanadate is attributable entirely to its suppression of feeding.

Induction of tolerance to islet xenografts in a concordant rat-to-mouse model.

ADP ribosylation by cholera toxin identifies three G-proteins that are activated by the galanin receptor. Studies with RINm5F cell membranes.

Isolation of nonobese diabetic mouse T-cells that recognize novel autoantigens involved in the early events of diabetes.

Abnormal insulin secretion, not insulin resistance, is the genetic or primary defect of MODY in the RW pedigree.

Peculiar T-cell signaling does not preclude positive selection in the diabetes-prone BB rat.

Biological actions of insulin are differentially regulated by glucose and insulin in primary cultured adipocytes. Chronic ability to increase glycogen synthase activity.

Maternal effect and familial aggregation in NIDDM. The CODIAB Study. CODIAB-INSERM-ZENECA Study Group.

Growth hormone induces resistance to the mitogenic action of insulin through local IGF-I. Studies in normal and Pygmy T-cell lines.

Effects of calcium antagonists on insulin-mediated glucose metabolism in skeletal muscle.

Autoantibodies to glutamic acid decarboxylase in patients with IDDM and autoimmune thyroid disease.

High genetic risk for IDDM in the Pacific Northwest. First report from the Washington State Diabetes Prediction Study.

Synthesis of type VI collagen by cultured glomerular cells and comparison of its regulation by glucose and other factors with that of type IV collagen.

Factors responsible for impaired fibrinolysis in obese subjects and NIDDM patients.

Major histocompatibility complex-encoded antigen processing gene polymorphism in IDDM.

Delayed onset of insulin activation of the insulin receptor kinase in vivo in human skeletal muscle.

Identification in islets of Langerhans of a new rat alpha 2-adrenergic receptor.

Predictive value of lymphocyte antibodies for the appearance of diabetes in BB rats.

A new transgenic mouse model of chronic hyperglycemia.

Associations of anti-GAD antibodies with islet cell antibodies and insulin autoantibodies in first-degree relatives of type I diabetic patients.

GAD autoantibodies in IDDM, stiff-man syndrome, and autoimmune polyendocrine syndrome type I recognize different epitopes.

Maternal component in NIDDM transmission. How large an effect?

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