Index by Author

June 1997; 46 (6)
  1. A
  2. B
  3. C
  4. D
  5. E
  6. F
  7. G
  8. H
  9. I
  10. J
  11. K
  12. L
  13. M
  14. N
  15. O
  16. P
  17. Q
  18. R
  19. S
  20. T
  21. U
  22. V
  23. W
  24. X
  25. Y
  26. Z

A

  1. Abrams-Carter, L

    • Regulation of glycogen synthase activity in cultured skeletal muscle cells from subjects with type II diabetes: role of chronic hyperinsulinemia and hyperglycemia. Diabetes June 1997 46:1017-1024; doi:10.2337/diabetes.46.6.1017

  2. Allander, S V

    • Analysis of candidate genes for susceptibility to type I diabetes: a case-control and family-association study of genes on chromosome 2q31-35. Diabetes June 1997 46:1069-1074; doi:10.2337/diabetes.46.6.1069

  3. Araki, E

    • Impact of natural IRS-1 mutations on insulin signals: mutations of IRS-1 in the PTB domain and near SH2 protein binding sites result in impaired function at different steps of IRS-1 signaling. Diabetes June 1997 46:929-936; doi:10.2337/diabetes.46.6.929

  4. Avogaro, A

    • Forearm nitric oxide balance, vascular relaxation, and glucose metabolism in NIDDM patients. Diabetes June 1997 46:1040-1046; doi:10.2337/diabetes.46.6.1040

  5. Aznar, J

    • Modulatory effect of erythrocytes on the platelet reactivity to collagen in IDDM patients. Diabetes June 1997 46:1047-1053; doi:10.2337/diabetes.46.6.1047

B

  1. Bailyes, E M

    • Characterization of the unusual insulin of Psammomys obesus, a rodent with nutrition-induced NIDDM-like syndrome. Diabetes June 1997 46:953-957; doi:10.2337/diabetes.46.6.953

  2. Baloga, J

    • Mechanisms of the kinetic defect in insulin action in obesity and NIDDM. Diabetes June 1997 46:994-1000; doi:10.2337/diabetes.46.6.994

  3. Barberá, G

    • Modulatory effect of erythrocytes on the platelet reactivity to collagen in IDDM patients. Diabetes June 1997 46:1047-1053; doi:10.2337/diabetes.46.6.1047

  4. Barker, C F

    • B-cells are required for the initiation of insulitis and sialitis in nonobese diabetic mice. Diabetes June 1997 46:941-946; doi:10.2337/diabetes.46.6.941

  5. Baur, L A

    • Skeletal muscle triglyceride levels are inversely related to insulin action. Diabetes June 1997 46:983-988; doi:10.2337/diabetes.46.6.983

  6. BeltrandelRio, H

    • Regulation of insulin secretion from novel engineered insulinoma cell lines. Diabetes June 1997 46:968-977; doi:10.2337/diabetes.46.6.968

  7. Berkemeier, L

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

  8. Bogardus, C

    • Skeletal muscle triglyceride levels are inversely related to insulin action. Diabetes June 1997 46:983-988; doi:10.2337/diabetes.46.6.983

  9. Bolli, G B

    • Evidence of increased systemic glucose production and gluconeogenesis in an early stage of NIDDM. Diabetes June 1997 46:1010-1016; doi:10.2337/diabetes.46.6.1010

  10. Bougnères, P F

    • Circulating leptin in normal children and during the dynamic phase of juvenile obesity: relation to body fatness, energy metabolism, caloric intake, and sexual dimorphism. Diabetes June 1997 46:989-993; doi:10.2337/diabetes.46.6.989

  11. Brosius, F C

    • D-glucose stimulates mesangial cell GLUT1 expression and basal and IGF-I-sensitive glucose uptake in rat mesangial cells: implications for diabetic nephropathy. Diabetes June 1997 46:1030-1039; doi:10.2337/diabetes.46.6.1030

  12. Brunetti, P

    • Evidence of increased systemic glucose production and gluconeogenesis in an early stage of NIDDM. Diabetes June 1997 46:1010-1016; doi:10.2337/diabetes.46.6.1010

C

  1. Cailleau, C

    • Treatment with neutralizing antibodies specific for IL-1beta prevents cyclophosphamide-induced diabetes in nonobese diabetic mice. Diabetes June 1997 46:937-940; doi:10.2337/diabetes.46.6.937

  2. Calveri, M

    • Forearm nitric oxide balance, vascular relaxation, and glucose metabolism in NIDDM patients. Diabetes June 1997 46:1040-1046; doi:10.2337/diabetes.46.6.1040

  3. Calò, L

    • Forearm nitric oxide balance, vascular relaxation, and glucose metabolism in NIDDM patients. Diabetes June 1997 46:1040-1046; doi:10.2337/diabetes.46.6.1040

  4. Carlsson, M

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

  5. Carlsson, P O

    • Islet capillary blood pressure increase mediated by hyperglycemia in NIDDM GK rats. Diabetes June 1997 46:947-952; doi:10.2337/diabetes.46.6.947

  6. Carmena, R

    • Modulatory effect of erythrocytes on the platelet reactivity to collagen in IDDM patients. Diabetes June 1997 46:1047-1053; doi:10.2337/diabetes.46.6.1047

  7. Carnaud, C

    • Treatment with neutralizing antibodies specific for IL-1beta prevents cyclophosphamide-induced diabetes in nonobese diabetic mice. Diabetes June 1997 46:937-940; doi:10.2337/diabetes.46.6.937

  8. Cerasi, E

    • Characterization of the unusual insulin of Psammomys obesus, a rodent with nutrition-induced NIDDM-like syndrome. Diabetes June 1997 46:953-957; doi:10.2337/diabetes.46.6.953

  9. Chiu, K C

    • Genetics of NIDDM in France: studies with 19 candidate genes in affected sib pairs. Diabetes June 1997 46:1062-1068; doi:10.2337/diabetes.46.6.1062

  10. Ciaraldi, T P

    • Regulation of glycogen synthase activity in cultured skeletal muscle cells from subjects with type II diabetes: role of chronic hyperinsulinemia and hyperglycemia. Diabetes June 1997 46:1017-1024; doi:10.2337/diabetes.46.6.1017

  11. Ciofetta, M

    • Evidence of increased systemic glucose production and gluconeogenesis in an early stage of NIDDM. Diabetes June 1997 46:1010-1016; doi:10.2337/diabetes.46.6.1010

  12. Clark, S A

    • Novel insulinoma cell lines produced by iterative engineering of GLUT2, glucokinase, and human insulin expression. Diabetes June 1997 46:958-967; doi:10.2337/diabetes.46.6.958
    • Regulation of insulin secretion from novel engineered insulinoma cell lines. Diabetes June 1997 46:968-977; doi:10.2337/diabetes.46.6.968

  13. Cobelli, C

    • Forearm nitric oxide balance, vascular relaxation, and glucose metabolism in NIDDM patients. Diabetes June 1997 46:1040-1046; doi:10.2337/diabetes.46.6.1040

  14. Concepcion, L A

    • D-glucose stimulates mesangial cell GLUT1 expression and basal and IGF-I-sensitive glucose uptake in rat mesangial cells: implications for diabetic nephropathy. Diabetes June 1997 46:1030-1039; doi:10.2337/diabetes.46.6.1030

  15. Constandy, H

    • Novel insulinoma cell lines produced by iterative engineering of GLUT2, glucokinase, and human insulin expression. Diabetes June 1997 46:958-967; doi:10.2337/diabetes.46.6.958

D

  1. De Boissieu, D

    • Circulating leptin in normal children and during the dynamic phase of juvenile obesity: relation to body fatness, energy metabolism, caloric intake, and sexual dimorphism. Diabetes June 1997 46:989-993; doi:10.2337/diabetes.46.6.989

  2. Del Prato, S

    • Forearm nitric oxide balance, vascular relaxation, and glucose metabolism in NIDDM patients. Diabetes June 1997 46:1040-1046; doi:10.2337/diabetes.46.6.1040

  3. Del Sindaco, P

    • Evidence of increased systemic glucose production and gluconeogenesis in an early stage of NIDDM. Diabetes June 1997 46:1010-1016; doi:10.2337/diabetes.46.6.1010

  4. Demenais, F

    • Genetics of NIDDM in France: studies with 19 candidate genes in affected sib pairs. Diabetes June 1997 46:1062-1068; doi:10.2337/diabetes.46.6.1062

  5. Diu-Hercend, A

    • Treatment with neutralizing antibodies specific for IL-1beta prevents cyclophosphamide-induced diabetes in nonobese diabetic mice. Diabetes June 1997 46:937-940; doi:10.2337/diabetes.46.6.937

  6. Duyk, G M

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

E

  1. El-Hashimy, M

    • Acute postchallenge hyperinsulinemia predicts weight gain: a prospective study. Diabetes June 1997 46:1025-1029; doi:10.2337/diabetes.46.6.1025

  2. England, R L

    • D-glucose stimulates mesangial cell GLUT1 expression and basal and IGF-I-sensitive glucose uptake in rat mesangial cells: implications for diabetic nephropathy. Diabetes June 1997 46:1030-1039; doi:10.2337/diabetes.46.6.1030

  3. Eriksson, U J

    • Vitamin E decreases the occurrence of malformations in the offspring of diabetic rats. Diabetes June 1997 46:1054-1061; doi:10.2337/diabetes.46.6.1054

F

  1. Fang, L

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

  2. Ferber, S

    • Novel insulinoma cell lines produced by iterative engineering of GLUT2, glucokinase, and human insulin expression. Diabetes June 1997 46:958-967; doi:10.2337/diabetes.46.6.958

  3. Forsblom, C M

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

  4. Freytag, S O

    • D-glucose stimulates mesangial cell GLUT1 expression and basal and IGF-I-sensitive glucose uptake in rat mesangial cells: implications for diabetic nephropathy. Diabetes June 1997 46:1030-1039; doi:10.2337/diabetes.46.6.1030

  5. Froguel, P

    • Genetics of NIDDM in France: studies with 19 candidate genes in affected sib pairs. Diabetes June 1997 46:1062-1068; doi:10.2337/diabetes.46.6.1062

  6. Furukawa, N

    • Impact of natural IRS-1 mutations on insulin signals: mutations of IRS-1 in the PTB domain and near SH2 protein binding sites result in impaired function at different steps of IRS-1 signaling. Diabetes June 1997 46:929-936; doi:10.2337/diabetes.46.6.929

G

  1. Gabbay, K H

    • Analysis of candidate genes for susceptibility to type I diabetes: a case-control and family-association study of genes on chromosome 2q31-35. Diabetes June 1997 46:1069-1074; doi:10.2337/diabetes.46.6.1069

  2. Gerow, K

    • Metabolic defects in lean nondiabetic offspring of NIDDM parents: a cross-sectional study. Diabetes June 1997 46:1001-1009; doi:10.2337/diabetes.46.6.1001

  3. Ghosh, S

    • Metabolic defects in lean nondiabetic offspring of NIDDM parents: a cross-sectional study. Diabetes June 1997 46:1001-1009; doi:10.2337/diabetes.46.6.1001

  4. Gilbert, J D

    • D-glucose stimulates mesangial cell GLUT1 expression and basal and IGF-I-sensitive glucose uptake in rat mesangial cells: implications for diabetic nephropathy. Diabetes June 1997 46:1030-1039; doi:10.2337/diabetes.46.6.1030

  5. Glaser, B

    • Genetics of NIDDM in France: studies with 19 candidate genes in affected sib pairs. Diabetes June 1997 46:1062-1068; doi:10.2337/diabetes.46.6.1062

  6. Glucksmann, M A

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

  7. Goranson, J

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

  8. Gross, D J

    • Characterization of the unusual insulin of Psammomys obesus, a rodent with nutrition-induced NIDDM-like syndrome. Diabetes June 1997 46:953-957; doi:10.2337/diabetes.46.6.953

H

  1. Habener, J F

    • Leptin suppression of insulin secretion by the activation of ATP-sensitive K+ channels in pancreatic beta-cells. Diabetes June 1997 46:1087-1093; doi:10.2337/diabetes.46.6.1087

  2. Hager, J

    • Genetics of NIDDM in France: studies with 19 candidate genes in affected sib pairs. Diabetes June 1997 46:1062-1068; doi:10.2337/diabetes.46.6.1062

  3. Halban, P

    • Novel insulinoma cell lines produced by iterative engineering of GLUT2, glucokinase, and human insulin expression. Diabetes June 1997 46:958-967; doi:10.2337/diabetes.46.6.958

  4. Halban, P A

    • Proinsulin conversion in GH3 cells after coexpression of human proinsulin with the endoproteases PC2 and/or PC3. Diabetes June 1997 46:978-982; doi:10.2337/diabetes.46.6.978

  5. Hani, E H

    • Genetics of NIDDM in France: studies with 19 candidate genes in affected sib pairs. Diabetes June 1997 46:1062-1068; doi:10.2337/diabetes.46.6.1062

  6. Hansen, B V

    • Polymorphisms in the interleukin-1 gene cluster do not contribute to the genetic susceptibility of diabetic nephropathy in Caucasian patients with IDDM. Diabetes June 1997 46:1075-1076

  7. Hansen, P

    • Novel insulinoma cell lines produced by iterative engineering of GLUT2, glucokinase, and human insulin expression. Diabetes June 1997 46:958-967; doi:10.2337/diabetes.46.6.958

  8. Hansen, P M

    • Polymorphisms in the interleukin-1 gene cluster do not contribute to the genetic susceptibility of diabetic nephropathy in Caucasian patients with IDDM. Diabetes June 1997 46:1075-1076

  9. Heilig, C W

    • D-glucose stimulates mesangial cell GLUT1 expression and basal and IGF-I-sensitive glucose uptake in rat mesangial cells: implications for diabetic nephropathy. Diabetes June 1997 46:1030-1039; doi:10.2337/diabetes.46.6.1030

  10. Heilig, K O

    • D-glucose stimulates mesangial cell GLUT1 expression and basal and IGF-I-sensitive glucose uptake in rat mesangial cells: implications for diabetic nephropathy. Diabetes June 1997 46:1030-1039; doi:10.2337/diabetes.46.6.1030

  11. Heller, R S

    • Leptin suppression of insulin secretion by the activation of ATP-sensitive K+ channels in pancreatic beta-cells. Diabetes June 1997 46:1087-1093; doi:10.2337/diabetes.46.6.1087

  12. Henkel-Rieger, R

    • Regulation of insulin secretion from novel engineered insulinoma cell lines. Diabetes June 1997 46:968-977; doi:10.2337/diabetes.46.6.968

  13. Henry, R R

    • Regulation of glycogen synthase activity in cultured skeletal muscle cells from subjects with type II diabetes: role of chronic hyperinsulinemia and hyperglycemia. Diabetes June 1997 46:1017-1024; doi:10.2337/diabetes.46.6.1017

  14. Hohmeier, H E

    • Regulation of insulin secretion from novel engineered insulinoma cell lines. Diabetes June 1997 46:968-977; doi:10.2337/diabetes.46.6.968

  15. Holz, G G

    • Leptin suppression of insulin secretion by the activation of ATP-sensitive K+ channels in pancreatic beta-cells. Diabetes June 1997 46:1087-1093; doi:10.2337/diabetes.46.6.1087

  16. Hutton, J C

    • Characterization of the unusual insulin of Psammomys obesus, a rodent with nutrition-induced NIDDM-like syndrome. Diabetes June 1997 46:953-957; doi:10.2337/diabetes.46.6.953

I

  1. Iida, M

    • Leptin receptor of Zucker fatty rat performs reduced signal transduction. Diabetes June 1997 46:1077-1080; doi:10.2337/diabetes.46.6.1077

  2. Irminger, J C

    • Proinsulin conversion in GH3 cells after coexpression of human proinsulin with the endoproteases PC2 and/or PC3. Diabetes June 1997 46:978-982; doi:10.2337/diabetes.46.6.978

J

  1. Jansson, L

    • Islet capillary blood pressure increase mediated by hyperglycemia in NIDDM GK rats. Diabetes June 1997 46:947-952; doi:10.2337/diabetes.46.6.947

  2. Jenkins, A B

    • Skeletal muscle triglyceride levels are inversely related to insulin action. Diabetes June 1997 46:983-988; doi:10.2337/diabetes.46.6.983

K

  1. Kaiser, N

    • Characterization of the unusual insulin of Psammomys obesus, a rodent with nutrition-induced NIDDM-like syndrome. Diabetes June 1997 46:953-957; doi:10.2337/diabetes.46.6.953

  2. Kaneko, K

    • Impact of natural IRS-1 mutations on insulin signals: mutations of IRS-1 in the PTB domain and near SH2 protein binding sites result in impaired function at different steps of IRS-1 signaling. Diabetes June 1997 46:929-936; doi:10.2337/diabetes.46.6.929

  3. Kaufmann, J E

    • Proinsulin conversion in GH3 cells after coexpression of human proinsulin with the endoproteases PC2 and/or PC3. Diabetes June 1997 46:978-982; doi:10.2337/diabetes.46.6.978

  4. Kennedy, G

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

  5. Kern, J

    • B-cells are required for the initiation of insulitis and sialitis in nonobese diabetic mice. Diabetes June 1997 46:941-946; doi:10.2337/diabetes.46.6.941

  6. Kieffer, T J

    • Leptin suppression of insulin secretion by the activation of ATP-sensitive K+ channels in pancreatic beta-cells. Diabetes June 1997 46:1087-1093; doi:10.2337/diabetes.46.6.1087

  7. Kishikawa, H

    • Impact of natural IRS-1 mutations on insulin signals: mutations of IRS-1 in the PTB domain and near SH2 protein binding sites result in impaired function at different steps of IRS-1 signaling. Diabetes June 1997 46:929-936; doi:10.2337/diabetes.46.6.929

  8. Kriketos, A D

    • Skeletal muscle triglyceride levels are inversely related to insulin action. Diabetes June 1997 46:983-988; doi:10.2337/diabetes.46.6.983

  9. Krolewski, A S

    • Acute postchallenge hyperinsulinemia predicts weight gain: a prospective study. Diabetes June 1997 46:1025-1029; doi:10.2337/diabetes.46.6.1025

  10. Kuwajima, M

    • Leptin receptor of Zucker fatty rat performs reduced signal transduction. Diabetes June 1997 46:1077-1080; doi:10.2337/diabetes.46.6.1077

  11. Källskog, O

    • Islet capillary blood pressure increase mediated by hyperglycemia in NIDDM GK rats. Diabetes June 1997 46:947-952; doi:10.2337/diabetes.46.6.947

L

  1. Lahlou, N

    • Circulating leptin in normal children and during the dynamic phase of juvenile obesity: relation to body fatness, energy metabolism, caloric intake, and sexual dimorphism. Diabetes June 1997 46:989-993; doi:10.2337/diabetes.46.6.989

  2. Lalli, C

    • Evidence of increased systemic glucose production and gluconeogenesis in an early stage of NIDDM. Diabetes June 1997 46:1010-1016; doi:10.2337/diabetes.46.6.1010

  3. Landais, P

    • Circulating leptin in normal children and during the dynamic phase of juvenile obesity: relation to body fatness, energy metabolism, caloric intake, and sexual dimorphism. Diabetes June 1997 46:989-993; doi:10.2337/diabetes.46.6.989

  4. Leech, C A

    • Leptin suppression of insulin secretion by the activation of ATP-sensitive K+ channels in pancreatic beta-cells. Diabetes June 1997 46:1087-1093; doi:10.2337/diabetes.46.6.1087

  5. Lehto, M

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

  6. Lesage, S

    • Genetics of NIDDM in France: studies with 19 candidate genes in affected sib pairs. Diabetes June 1997 46:1062-1068; doi:10.2337/diabetes.46.6.1062

  7. Lillioja, S

    • Skeletal muscle triglyceride levels are inversely related to insulin action. Diabetes June 1997 46:983-988; doi:10.2337/diabetes.46.6.983

  8. Liu, Y

    • D-glucose stimulates mesangial cell GLUT1 expression and basal and IGF-I-sensitive glucose uptake in rat mesangial cells: implications for diabetic nephropathy. Diabetes June 1997 46:1030-1039; doi:10.2337/diabetes.46.6.1030

  9. Ludvik, B

    • Mechanisms of the kinetic defect in insulin action in obesity and NIDDM. Diabetes June 1997 46:994-1000; doi:10.2337/diabetes.46.6.994

M

  1. Markel, P

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

  2. Martin, B C

    • Acute postchallenge hyperinsulinemia predicts weight gain: a prospective study. Diabetes June 1997 46:1025-1029; doi:10.2337/diabetes.46.6.1025

  3. Matsuda, K

    • Impact of natural IRS-1 mutations on insulin signals: mutations of IRS-1 in the PTB domain and near SH2 protein binding sites result in impaired function at different steps of IRS-1 signaling. Diabetes June 1997 46:929-936; doi:10.2337/diabetes.46.6.929

  4. McMenimen, C

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

  5. Milner, M R

    • Skeletal muscle triglyceride levels are inversely related to insulin action. Diabetes June 1997 46:983-988; doi:10.2337/diabetes.46.6.983

  6. Miola, M

    • Forearm nitric oxide balance, vascular relaxation, and glucose metabolism in NIDDM patients. Diabetes June 1997 46:1040-1046; doi:10.2337/diabetes.46.6.1040

  7. Motoshima, H

    • Impact of natural IRS-1 mutations on insulin signals: mutations of IRS-1 in the PTB domain and near SH2 protein binding sites result in impaired function at different steps of IRS-1 signaling. Diabetes June 1997 46:929-936; doi:10.2337/diabetes.46.6.929

  8. Mudaliar, S

    • Regulation of glycogen synthase activity in cultured skeletal muscle cells from subjects with type II diabetes: role of chronic hyperinsulinemia and hyperglycemia. Diabetes June 1997 46:1017-1024; doi:10.2337/diabetes.46.6.1017

  9. Mungall, J

    • Proinsulin conversion in GH3 cells after coexpression of human proinsulin with the endoproteases PC2 and/or PC3. Diabetes June 1997 46:978-982; doi:10.2337/diabetes.46.6.978

  10. Munnelly, K D

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

  11. Murakami, T

    • Leptin receptor of Zucker fatty rat performs reduced signal transduction. Diabetes June 1997 46:1077-1080; doi:10.2337/diabetes.46.6.1077

N

  1. Naji, A

    • B-cells are required for the initiation of insulitis and sialitis in nonobese diabetic mice. Diabetes June 1997 46:941-946; doi:10.2337/diabetes.46.6.941

  2. Naya, F J

    • Analysis of candidate genes for susceptibility to type I diabetes: a case-control and family-association study of genes on chromosome 2q31-35. Diabetes June 1997 46:1069-1074; doi:10.2337/diabetes.46.6.1069

  3. Newgard, C B

    • Novel insulinoma cell lines produced by iterative engineering of GLUT2, glucokinase, and human insulin expression. Diabetes June 1997 46:958-967; doi:10.2337/diabetes.46.6.958
    • Regulation of insulin secretion from novel engineered insulinoma cell lines. Diabetes June 1997 46:968-977; doi:10.2337/diabetes.46.6.968

  4. Nielsen, F S

    • Polymorphisms in the interleukin-1 gene cluster do not contribute to the genetic susceptibility of diabetic nephropathy in Caucasian patients with IDDM. Diabetes June 1997 46:1075-1076

  5. Nikoulina, S E

    • Regulation of glycogen synthase activity in cultured skeletal muscle cells from subjects with type II diabetes: role of chronic hyperinsulinemia and hyperglycemia. Diabetes June 1997 46:1017-1024; doi:10.2337/diabetes.46.6.1017

  6. Nir, W J

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

  7. Nolan, J J

    • Mechanisms of the kinetic defect in insulin action in obesity and NIDDM. Diabetes June 1997 46:994-1000; doi:10.2337/diabetes.46.6.994

  8. Noorchashm, H

    • B-cells are required for the initiation of insulitis and sialitis in nonobese diabetic mice. Diabetes June 1997 46:941-946; doi:10.2337/diabetes.46.6.941

  9. Noorchashm, N

    • B-cells are required for the initiation of insulitis and sialitis in nonobese diabetic mice. Diabetes June 1997 46:941-946; doi:10.2337/diabetes.46.6.941

  10. Normington, K

    • Novel insulinoma cell lines produced by iterative engineering of GLUT2, glucokinase, and human insulin expression. Diabetes June 1997 46:958-967; doi:10.2337/diabetes.46.6.958
    • Regulation of insulin secretion from novel engineered insulinoma cell lines. Diabetes June 1997 46:968-977; doi:10.2337/diabetes.46.6.968

O

  1. Olefsky, J M

    • Mechanisms of the kinetic defect in insulin action in obesity and NIDDM. Diabetes June 1997 46:994-1000; doi:10.2337/diabetes.46.6.994

  2. Orho, M

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

  3. Ostenson, C G

    • Islet capillary blood pressure increase mediated by hyperglycemia in NIDDM GK rats. Diabetes June 1997 46:947-952; doi:10.2337/diabetes.46.6.947

  4. Owerbach, D

    • Analysis of candidate genes for susceptibility to type I diabetes: a case-control and family-association study of genes on chromosome 2q31-35. Diabetes June 1997 46:1069-1074; doi:10.2337/diabetes.46.6.1069

P

  1. Pampanelli, S

    • Evidence of increased systemic glucose production and gluconeogenesis in an early stage of NIDDM. Diabetes June 1997 46:1010-1016; doi:10.2337/diabetes.46.6.1010

  2. Pan, D A

    • Skeletal muscle triglyceride levels are inversely related to insulin action. Diabetes June 1997 46:983-988; doi:10.2337/diabetes.46.6.983

  3. Park, K S

    • Regulation of glycogen synthase activity in cultured skeletal muscle cells from subjects with type II diabetes: role of chronic hyperinsulinemia and hyperglycemia. Diabetes June 1997 46:1017-1024; doi:10.2337/diabetes.46.6.1017

  4. Parving, H H

    • Polymorphisms in the interleukin-1 gene cluster do not contribute to the genetic susceptibility of diabetic nephropathy in Caucasian patients with IDDM. Diabetes June 1997 46:1075-1076

  5. Passa, P

    • Genetics of NIDDM in France: studies with 19 candidate genes in affected sib pairs. Diabetes June 1997 46:1062-1068; doi:10.2337/diabetes.46.6.1062

  6. Pavan, P

    • Forearm nitric oxide balance, vascular relaxation, and glucose metabolism in NIDDM patients. Diabetes June 1997 46:1040-1046; doi:10.2337/diabetes.46.6.1040

  7. Permutt, M A

    • Genetics of NIDDM in France: studies with 19 candidate genes in affected sib pairs. Diabetes June 1997 46:1062-1068; doi:10.2337/diabetes.46.6.1062

  8. Perriello, G

    • Evidence of increased systemic glucose production and gluconeogenesis in an early stage of NIDDM. Diabetes June 1997 46:1010-1016; doi:10.2337/diabetes.46.6.1010

  9. Perseghin, G

    • Metabolic defects in lean nondiabetic offspring of NIDDM parents: a cross-sectional study. Diabetes June 1997 46:1001-1009; doi:10.2337/diabetes.46.6.1001

  10. Philippi, A

    • Genetics of NIDDM in France: studies with 19 candidate genes in affected sib pairs. Diabetes June 1997 46:1062-1068; doi:10.2337/diabetes.46.6.1062

  11. Piarulli, F

    • Forearm nitric oxide balance, vascular relaxation, and glucose metabolism in NIDDM patients. Diabetes June 1997 46:1040-1046; doi:10.2337/diabetes.46.6.1040

  12. Pociot, F

    • Polymorphisms in the interleukin-1 gene cluster do not contribute to the genetic susceptibility of diabetic nephropathy in Caucasian patients with IDDM. Diabetes June 1997 46:1075-1076

  13. Powell, D R

    • Analysis of candidate genes for susceptibility to type I diabetes: a case-control and family-association study of genes on chromosome 2q31-35. Diabetes June 1997 46:1069-1074; doi:10.2337/diabetes.46.6.1069

  14. Pulido, J C

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

Q

  1. Quaade, C

    • Novel insulinoma cell lines produced by iterative engineering of GLUT2, glucokinase, and human insulin expression. Diabetes June 1997 46:958-967; doi:10.2337/diabetes.46.6.958

R

  1. Reichart, D

    • Mechanisms of the kinetic defect in insulin action in obesity and NIDDM. Diabetes June 1997 46:994-1000; doi:10.2337/diabetes.46.6.994

  2. Rosenzweig, J

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

  3. Rossing, P

    • Polymorphisms in the interleukin-1 gene cluster do not contribute to the genetic susceptibility of diabetic nephropathy in Caucasian patients with IDDM. Diabetes June 1997 46:1075-1076

  4. Rostami, S Y

    • B-cells are required for the initiation of insulitis and sialitis in nonobese diabetic mice. Diabetes June 1997 46:941-946; doi:10.2337/diabetes.46.6.941

  5. Ruuth, E

    • Treatment with neutralizing antibodies specific for IL-1beta prevents cyclophosphamide-induced diabetes in nonobese diabetic mice. Diabetes June 1997 46:937-940; doi:10.2337/diabetes.46.6.937

S

  1. Santeusanio, F

    • Evidence of increased systemic glucose production and gluconeogenesis in an early stage of NIDDM. Diabetes June 1997 46:1010-1016; doi:10.2337/diabetes.46.6.1010

  2. Santos, M T

    • Modulatory effect of erythrocytes on the platelet reactivity to collagen in IDDM patients. Diabetes June 1997 46:1047-1053; doi:10.2337/diabetes.46.6.1047

  3. Schneider, B S

    • Characterization of the unusual insulin of Psammomys obesus, a rodent with nutrition-induced NIDDM-like syndrome. Diabetes June 1997 46:953-957; doi:10.2337/diabetes.46.6.953

  4. Scotti, S

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

  5. Shichiri, M

    • Impact of natural IRS-1 mutations on insulin signals: mutations of IRS-1 in the PTB domain and near SH2 protein binding sites result in impaired function at different steps of IRS-1 signaling. Diabetes June 1997 46:929-936; doi:10.2337/diabetes.46.6.929

  6. Shima, K

    • Leptin receptor of Zucker fatty rat performs reduced signal transduction. Diabetes June 1997 46:1077-1080; doi:10.2337/diabetes.46.6.1077

  7. Shulman, G I

    • Metabolic defects in lean nondiabetic offspring of NIDDM parents: a cross-sectional study. Diabetes June 1997 46:1001-1009; doi:10.2337/diabetes.46.6.1001

  8. Sigal, R J

    • Acute postchallenge hyperinsulinemia predicts weight gain: a prospective study. Diabetes June 1997 46:1025-1029; doi:10.2337/diabetes.46.6.1025

  9. Simán, C M

    • Vitamin E decreases the occurrence of malformations in the offspring of diabetic rats. Diabetes June 1997 46:1054-1061; doi:10.2337/diabetes.46.6.1054

  10. Soeldner, J S

    • Acute postchallenge hyperinsulinemia predicts weight gain: a prospective study. Diabetes June 1997 46:1025-1029; doi:10.2337/diabetes.46.6.1025

  11. Steiner, D F

    • Characterization of the unusual insulin of Psammomys obesus, a rodent with nutrition-induced NIDDM-like syndrome. Diabetes June 1997 46:953-957; doi:10.2337/diabetes.46.6.953

  12. Stoffel, M

    • Genetics of NIDDM in France: studies with 19 candidate genes in affected sib pairs. Diabetes June 1997 46:1062-1068; doi:10.2337/diabetes.46.6.1062

  13. Storlien, L H

    • Skeletal muscle triglyceride levels are inversely related to insulin action. Diabetes June 1997 46:983-988; doi:10.2337/diabetes.46.6.983

T

  1. Tanizawa, Y

    • Genetics of NIDDM in France: studies with 19 candidate genes in affected sib pairs. Diabetes June 1997 46:1062-1068; doi:10.2337/diabetes.46.6.1062

  2. Tarnow, L

    • Polymorphisms in the interleukin-1 gene cluster do not contribute to the genetic susceptibility of diabetic nephropathy in Caucasian patients with IDDM. Diabetes June 1997 46:1075-1076

  3. Tayber, O

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

  4. Thomas, J D

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

  5. Tiengo, A

    • Forearm nitric oxide balance, vascular relaxation, and glucose metabolism in NIDDM patients. Diabetes June 1997 46:1040-1046; doi:10.2337/diabetes.46.6.1040

  6. Todaka, M

    • Impact of natural IRS-1 mutations on insulin signals: mutations of IRS-1 in the PTB domain and near SH2 protein binding sites result in impaired function at different steps of IRS-1 signaling. Diabetes June 1997 46:929-936; doi:10.2337/diabetes.46.6.929

  7. Tsai, M J

    • Analysis of candidate genes for susceptibility to type I diabetes: a case-control and family-association study of genes on chromosome 2q31-35. Diabetes June 1997 46:1069-1074; doi:10.2337/diabetes.46.6.1069

  8. Tsuruzoe, K

    • Impact of natural IRS-1 mutations on insulin signals: mutations of IRS-1 in the PTB domain and near SH2 protein binding sites result in impaired function at different steps of IRS-1 signaling. Diabetes June 1997 46:929-936; doi:10.2337/diabetes.46.6.929

  9. Tuomi, T

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

U

  1. Ura, S

    • Impact of natural IRS-1 mutations on insulin signals: mutations of IRS-1 in the PTB domain and near SH2 protein binding sites result in impaired function at different steps of IRS-1 signaling. Diabetes June 1997 46:929-936; doi:10.2337/diabetes.46.6.929

V

  1. Valerio, A

    • Forearm nitric oxide balance, vascular relaxation, and glucose metabolism in NIDDM patients. Diabetes June 1997 46:1040-1046; doi:10.2337/diabetes.46.6.1040

  2. Vallés, J

    • Modulatory effect of erythrocytes on the platelet reactivity to collagen in IDDM patients. Diabetes June 1997 46:1047-1053; doi:10.2337/diabetes.46.6.1047

  3. Varret, M

    • Genetics of NIDDM in France: studies with 19 candidate genes in affected sib pairs. Diabetes June 1997 46:1062-1068; doi:10.2337/diabetes.46.6.1062

  4. Velert, M

    • Modulatory effect of erythrocytes on the platelet reactivity to collagen in IDDM patients. Diabetes June 1997 46:1047-1053; doi:10.2337/diabetes.46.6.1047

  5. Vicini, P

    • Forearm nitric oxide balance, vascular relaxation, and glucose metabolism in NIDDM patients. Diabetes June 1997 46:1040-1046; doi:10.2337/diabetes.46.6.1040

  6. Vionnet, N

    • Genetics of NIDDM in France: studies with 19 candidate genes in affected sib pairs. Diabetes June 1997 46:1062-1068; doi:10.2337/diabetes.46.6.1062

  7. Volpi, E

    • Evidence of increased systemic glucose production and gluconeogenesis in an early stage of NIDDM. Diabetes June 1997 46:1010-1016; doi:10.2337/diabetes.46.6.1010

W

  1. Wantman, M

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

  2. Warram, J

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

  3. Warram, J H

    • Acute postchallenge hyperinsulinemia predicts weight gain: a prospective study. Diabetes June 1997 46:1025-1029; doi:10.2337/diabetes.46.6.1025

  4. Westwood, R

    • Treatment with neutralizing antibodies specific for IL-1beta prevents cyclophosphamide-induced diabetes in nonobese diabetic mice. Diabetes June 1997 46:937-940; doi:10.2337/diabetes.46.6.937

  5. Whitacre, J L

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

  6. Wu, Y

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

Y

  1. Yamashita, T

    • Leptin receptor of Zucker fatty rat performs reduced signal transduction. Diabetes June 1997 46:1077-1080; doi:10.2337/diabetes.46.6.1077

  2. Yoshimura, R

    • Impact of natural IRS-1 mutations on insulin signals: mutations of IRS-1 in the PTB domain and near SH2 protein binding sites result in impaired function at different steps of IRS-1 signaling. Diabetes June 1997 46:929-936; doi:10.2337/diabetes.46.6.929

  3. Yoshizato, K

    • Impact of natural IRS-1 mutations on insulin signals: mutations of IRS-1 in the PTB domain and near SH2 protein binding sites result in impaired function at different steps of IRS-1 signaling. Diabetes June 1997 46:929-936; doi:10.2337/diabetes.46.6.929

  4. Young, B M

    • Novel mutations and a mutational hotspot in the MODY3 gene. Diabetes June 1997 46:1081-1086; doi:10.2337/diabetes.46.6.1081

Z

  1. Zhu, M

    • D-glucose stimulates mesangial cell GLUT1 expression and basal and IGF-I-sensitive glucose uptake in rat mesangial cells: implications for diabetic nephropathy. Diabetes June 1997 46:1030-1039; doi:10.2337/diabetes.46.6.1030

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