Table of Contents

- P E Cryer
We are what we repeatedly do.
Diabetes February 1998 47:155-158
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- D J Drucker
Glucagon-like peptides.
Diabetes February 1998 47:159-169; doi:10.2337/diabetes.47.2.159
- Abstract
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- A G Nerlich,
- U Sauer,
- V Kolm-Litty,
- E Wagner,
- M Koch,
- and E D Schleicher
Expression of glutamine:fructose-6-phosphate amidotransferase in human tissues: evidence for high variability and distinct regulation in diabetes.
Diabetes February 1998 47:170-178; doi:10.2337/diabetes.47.2.170
- Abstract
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- R W Stevenson,
- D K Kreutter,
- K M Andrews,
- P E Genereux,
- and E M Gibbs
Possibility of distinct insulin-signaling pathways beyond phosphatidylinositol 3-kinase-mediating glucose transport and lipogenesis.
Diabetes February 1998 47:179-185; doi:10.2337/diabetes.47.2.179
- Abstract
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- D O Sobel,
- B Yankelevich,
- D Goyal,
- D Nelson,
- and A Mazumder
The B-subunit of cholera toxin induces immunoregulatory cells and prevents diabetes in the NOD mouse.
Diabetes February 1998 47:186-191; doi:10.2337/diabetes.47.2.186
- Abstract
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- V Nacher,
- J F Merino,
- M Raurell,
- J Soler,
- and E Montanya
Normoglycemia restores beta-cell replicative response to glucose in transplanted islets exposed to chronic hyperglycemia.
Diabetes February 1998 47:192-196; doi:10.2337/diabetes.47.2.192
- Abstract
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- A Lau,
- S Ramanathan,
- and P Poussier
Excessive production of nitric oxide by macrophages from DP-BB rats is secondary to the T-lymphopenic state of these animals.
Diabetes February 1998 47:197-205; doi:10.2337/diabetes.47.2.197
- Abstract
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- Y Sauvaire,
- P Petit,
- C Broca,
- M Manteghetti,
- Y Baissac,
- J Fernandez-Alvarez,
- R Gross,
- M Roye,
- A Leconte,
- R Gomis,
- and G Ribes
4-Hydroxyisoleucine: a novel amino acid potentiator of insulin secretion.
Diabetes February 1998 47:206-210; doi:10.2337/diabetes.47.2.206
- Abstract
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- I B Efanova,
- S V Zaitsev,
- G Brown,
- P O Berggren,
- and S Efendić
RX871024 induces Ca2+ mobilization from thapsigargin-sensitive stores in mouse pancreatic beta-cells.
Diabetes February 1998 47:211-218; doi:10.2337/diabetes.47.2.211
- Abstract
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- M Ookuma,
- K Ookuma,
- and D A York
Effects of leptin on insulin secretion from isolated rat pancreatic islets.
Diabetes February 1998 47:219-223; doi:10.2337/diabetes.47.2.219
- Abstract
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- P Masiello,
- C Broca,
- R Gross,
- M Roye,
- M Manteghetti,
- D Hillaire-Buys,
- M Novelli,
- and G Ribes
Experimental NIDDM: development of a new model in adult rats administered streptozotocin and nicotinamide.
Diabetes February 1998 47:224-229; doi:10.2337/diabetes.47.2.224
- Abstract
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- C S Mantzoros,
- R C Frederich,
- D Qu,
- B B Lowell,
- E Maratos-Flier,
- and J S Flier
Severe leptin resistance in brown fat-deficient uncoupling protein promoter-driven diphtheria toxin A mice despite suppression of hypothalamic neuropeptide Y and circulating corticosterone concentrations.
Diabetes February 1998 47:230-238; doi:10.2337/diabetes.47.2.230
- Abstract
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- S D Chessler,
- W Y Fujimoto,
- J B Shofer,
- E J Boyko,
- and D S Weigle
Increased plasma leptin levels are associated with fat accumulation in Japanese Americans.
Diabetes February 1998 47:239-243; doi:10.2337/diabetes.47.2.239
- Abstract
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- M Carantoni,
- F Abbasi,
- S Azhar,
- Y D Chen,
- M Klebanov,
- P W Wang,
- F Warmerdam,
- and G M Reaven
Plasma leptin concentrations do not appear to decrease insulin-mediated glucose disposal or glucose-stimulated insulin secretion in women with normal glucose tolerance.
Diabetes February 1998 47:244-247; doi:10.2337/diabetes.47.2.244
- Abstract
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- O P Pitkänen,
- P Nuutila,
- O T Raitakari,
- T Rönnemaa,
- P J Koskinen,
- H Iida,
- T J Lehtimäki,
- H K Laine,
- T Takala,
- J S Viikari,
- and J Knuuti
Coronary flow reserve is reduced in young men with IDDM.
Diabetes February 1998 47:248-254; doi:10.2337/diabetes.47.2.248
- Abstract
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- H Forsberg,
- U J Eriksson,
- O Melefors,
- and N Welsh
Beta-hydroxybutyrate increases reactive oxygen species in late but not in early postimplantation embryonic cells in vitro.
Diabetes February 1998 47:255-262; doi:10.2337/diabetes.47.2.255
- Abstract
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- P Hanifi Moghaddam,
- P de Knijf,
- B O Roep,
- B Van der Auwera,
- A Naipal,
- F Gorus,
- F Schuit,
- and M J Giphart
Genetic structure of IDDM1: two separate regions in the major histocompatibility complex contribute to susceptibility or protection. Belgian Diabetes Registry.
Diabetes February 1998 47:263-269; doi:10.2337/diabetes.47.2.263
- Abstract
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- J T Salonen,
- T A Lakka,
- H M Lakka,
- V P Valkonen,
- S A Everson,
- and G A Kaplan
Hyperinsulinemia is associated with the incidence of hypertension and dyslipidemia in middle-aged men.
Diabetes February 1998 47:270-275; doi:10.2337/diabetes.47.2.270
- Abstract
- M Tawata,
- M Ohtaka,
- E Iwase,
- Y Ikegishi,
- K Aida,
- and T Onaya
New mitochondrial DNA homoplasmic mutations associated with Japanese patients with type 2 diabetes.
Diabetes February 1998 47:276-277
- T A Chowdhury,
- P H Dyer,
- S Kumar,
- S P Gibson,
- B R Rowe,
- S J Davies,
- S M Marshall,
- P J Morris,
- G V Gill,
- S Feeney,
- P Maxwell,
- D Savage,
- A J Boulton,
- J A Todd,
- D Dunger,
- A H Barnett,
- and S C Bain
Association of apolipoprotein epsilon2 allele with diabetic nephropathy in Caucasian subjects with IDDM.
Diabetes February 1998 47:278-280
- O P Kristiansen,
- M Zamani,
- J Johannesen,
- T Mandrup-Poulsen,
- J J Cassiman,
- J Nerup,
- and F Pociot
Linkage and association between a CD4 gene polymorphism and IDDM in Danish IDDM patients. The Danish IDDM Epidemiology and Genetics Group, and The Danish Study Group of Diabetes in Childhood.
Diabetes February 1998 47:281-283
- J Magré,
- H Laurell,
- C Fizames,
- P J Antoine,
- C Dib,
- C Vigouroux,
- C Bourut,
- J Capeau,
- J Weissenbach,
- and D Langin
Human hormone-sensitive lipase: genetic mapping, identification of a new dinucleotide repeat, and association with obesity and NIDDM.
Diabetes February 1998 47:284-286
- K Kalidas,
- E Dow,
- P J Saker,
- N Wareham,
- D Halsall,
- R S Jackson,
- S P Chan,
- S Gelding,
- M Walker,
- E Kousta,
- D G Johnston,
- S O'Rahilly,
- and M I McCarthy
Prohormone convertase 1 in obesity, gestational diabetes mellitus, and NIDDM: no evidence for a major susceptibility role.
Diabetes February 1998 47:287-289
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- J Calles-Escandon,
- S A Mirza,
- B E Sobel,
- and D J Schneider
Induction of hyperinsulinemia combined with hyperglycemia and hypertriglyceridemia increases plasminogen activator inhibitor 1 in blood in normal human subjects.
Diabetes February 1998 47:290-293; doi:10.2337/diabetes.47.2.290
- Abstract
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- T M Mizuno,
- S P Kleopoulos,
- H T Bergen,
- J L Roberts,
- C A Priest,
- and C V Mobbs
Hypothalamic pro-opiomelanocortin mRNA is reduced by fasting and [corrected] in ob/ob and db/db mice, but is stimulated by leptin.
Diabetes February 1998 47:294-297; doi:10.2337/diabetes.47.2.294
- Abstract
Select this article
- D S Weigle,
- L E Selfridge,
- M W Schwartz,
- R J Seeley,
- D E Cummings,
- P J Havel,
- J L Kuijper,
- and H BeltrandelRio
Elevated free fatty acids induce uncoupling protein 3 expression in muscle: a potential explanation for the effect of fasting.
Diabetes February 1998 47:298-302; doi:10.2337/diabetes.47.2.298
- Abstract

Diabetes

We are what we repeatedly do.

Glucagon-like peptides.

Expression of glutamine:fructose-6-phosphate amidotransferase in human tissues: evidence for high variability and distinct regulation in diabetes.

Possibility of distinct insulin-signaling pathways beyond phosphatidylinositol 3-kinase-mediating glucose transport and lipogenesis.

The B-subunit of cholera toxin induces immunoregulatory cells and prevents diabetes in the NOD mouse.

Normoglycemia restores beta-cell replicative response to glucose in transplanted islets exposed to chronic hyperglycemia.

Excessive production of nitric oxide by macrophages from DP-BB rats is secondary to the T-lymphopenic state of these animals.

4-Hydroxyisoleucine: a novel amino acid potentiator of insulin secretion.

RX871024 induces Ca2+ mobilization from thapsigargin-sensitive stores in mouse pancreatic beta-cells.

Effects of leptin on insulin secretion from isolated rat pancreatic islets.

Experimental NIDDM: development of a new model in adult rats administered streptozotocin and nicotinamide.

Severe leptin resistance in brown fat-deficient uncoupling protein promoter-driven diphtheria toxin A mice despite suppression of hypothalamic neuropeptide Y and circulating corticosterone concentrations.

Increased plasma leptin levels are associated with fat accumulation in Japanese Americans.

Plasma leptin concentrations do not appear to decrease insulin-mediated glucose disposal or glucose-stimulated insulin secretion in women with normal glucose tolerance.

Coronary flow reserve is reduced in young men with IDDM.

Beta-hydroxybutyrate increases reactive oxygen species in late but not in early postimplantation embryonic cells in vitro.

Genetic structure of IDDM1: two separate regions in the major histocompatibility complex contribute to susceptibility or protection. Belgian Diabetes Registry.

Hyperinsulinemia is associated with the incidence of hypertension and dyslipidemia in middle-aged men.

New mitochondrial DNA homoplasmic mutations associated with Japanese patients with type 2 diabetes.

Association of apolipoprotein epsilon2 allele with diabetic nephropathy in Caucasian subjects with IDDM.

Linkage and association between a CD4 gene polymorphism and IDDM in Danish IDDM patients. The Danish IDDM Epidemiology and Genetics Group, and The Danish Study Group of Diabetes in Childhood.

Human hormone-sensitive lipase: genetic mapping, identification of a new dinucleotide repeat, and association with obesity and NIDDM.

Prohormone convertase 1 in obesity, gestational diabetes mellitus, and NIDDM: no evidence for a major susceptibility role.

Induction of hyperinsulinemia combined with hyperglycemia and hypertriglyceridemia increases plasminogen activator inhibitor 1 in blood in normal human subjects.

Hypothalamic pro-opiomelanocortin mRNA is reduced by fasting and [corrected] in ob/ob and db/db mice, but is stimulated by leptin.

Elevated free fatty acids induce uncoupling protein 3 expression in muscle: a potential explanation for the effect of fasting.

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