Glucose metabolism and insulin sensitivity in transgenic mice overexpressing leptin with lethal yellow agouti mutation: usefulness
of leptin for the treatment of obesity-associated diabetes. Diabetes August 1999 48:1615-1622; doi:10.2337/diabetes.48.8.1615
Augmentation of Ca2+-stimulated insulin release by glucose and long-chain fatty acids in rat pancreatic islets: free fatty
acids mimic ATP-sensitive K+ channel-independent insulinotropic action of glucose. Diabetes August 1999 48:1543-1549; doi:10.2337/diabetes.48.8.1543
Characterization of glucokinase mutations associated with maturity-onset diabetes of the young type 2 (MODY-2): different
glucokinase defects lead to a common phenotype. Diabetes August 1999 48:1645-1651; doi:10.2337/diabetes.48.8.1645
Dehydroepiandrosterone suppresses the elevated hepatic glucose-6-phosphatase and fructose-1,6-bisphosphatase activities in
C57BL/Ksj-db/db mice: comparison with troglitazone. Diabetes August 1999 48:1579-1585; doi:10.2337/diabetes.48.8.1579
Intramyocellular triglyceride content is a determinant of in vivo insulin resistance in humans: a 1H-13C nuclear magnetic
resonance spectroscopy assessment in offspring of type 2 diabetic parents. Diabetes August 1999 48:1600-1606; doi:10.2337/diabetes.48.8.1600
Hyperinsulinism caused by paternal-specific inheritance of a recessive mutation in the sulfonylurea-receptor gene. Diabetes August 1999 48:1652-1657; doi:10.2337/diabetes.48.8.1652
Hyperinsulinism caused by paternal-specific inheritance of a recessive mutation in the sulfonylurea-receptor gene. Diabetes August 1999 48:1652-1657; doi:10.2337/diabetes.48.8.1652
Intramyocellular triglyceride content is a determinant of in vivo insulin resistance in humans: a 1H-13C nuclear magnetic
resonance spectroscopy assessment in offspring of type 2 diabetic parents. Diabetes August 1999 48:1600-1606; doi:10.2337/diabetes.48.8.1600
Characterization of glucokinase mutations associated with maturity-onset diabetes of the young type 2 (MODY-2): different
glucokinase defects lead to a common phenotype. Diabetes August 1999 48:1645-1651; doi:10.2337/diabetes.48.8.1645
Relationship between insulin resistance and nonmodulating hypertension: linkage of metabolic abnormalities and cardiovascular
risk. Diabetes August 1999 48:1623-1630; doi:10.2337/diabetes.48.8.1623
Metabolic factors contributing to increased resting metabolic rate and decreased insulin-induced thermogenesis during the
development of type 2 diabetes. Diabetes August 1999 48:1607-1614; doi:10.2337/diabetes.48.8.1607
Mechanism responsible for inactivation of skeletal muscle pyruvate dehydrogenase complex in starvation and diabetes. Diabetes August 1999 48:1593-1599; doi:10.2337/diabetes.48.8.1593
Deficiency of phosphofructo-1-kinase/muscle subtype in humans is associated with impairment of insulin secretory oscillations.
Diabetes August 1999 48:1557-1561; doi:10.2337/diabetes.48.8.1557
Cellular immune response to phogrin in the NOD mouse: cloned T-cells cause destruction of islet transplants. Diabetes August 1999 48:1529-1534; doi:10.2337/diabetes.48.8.1529
Intramyocellular triglyceride content is a determinant of in vivo insulin resistance in humans: a 1H-13C nuclear magnetic
resonance spectroscopy assessment in offspring of type 2 diabetic parents. Diabetes August 1999 48:1600-1606; doi:10.2337/diabetes.48.8.1600
Intramyocellular triglyceride content is a determinant of in vivo insulin resistance in humans: a 1H-13C nuclear magnetic
resonance spectroscopy assessment in offspring of type 2 diabetic parents. Diabetes August 1999 48:1600-1606; doi:10.2337/diabetes.48.8.1600
Relationship between insulin resistance and nonmodulating hypertension: linkage of metabolic abnormalities and cardiovascular
risk. Diabetes August 1999 48:1623-1630; doi:10.2337/diabetes.48.8.1623
Relationship between insulin resistance and nonmodulating hypertension: linkage of metabolic abnormalities and cardiovascular
risk. Diabetes August 1999 48:1623-1630; doi:10.2337/diabetes.48.8.1623
Hyperinsulinism caused by paternal-specific inheritance of a recessive mutation in the sulfonylurea-receptor gene. Diabetes August 1999 48:1652-1657; doi:10.2337/diabetes.48.8.1652
Plasma phospholipid transfer protein activity is lowered by 24-h insulin and acipimox administration: blunted response to
insulin in type 2 diabetic patients. Diabetes August 1999 48:1631-1637; doi:10.2337/diabetes.48.8.1631
Glucose metabolism and insulin sensitivity in transgenic mice overexpressing leptin with lethal yellow agouti mutation: usefulness
of leptin for the treatment of obesity-associated diabetes. Diabetes August 1999 48:1615-1622; doi:10.2337/diabetes.48.8.1615
Glucose turnover and adipose tissue lipolysis are insulin-resistant in healthy relatives of type 2 diabetes patients: is cellular
insulin resistance a secondary phenomenon? Diabetes August 1999 48:1572-1578; doi:10.2337/diabetes.48.8.1572
Insulin-regulated mitochondrial gene expression is associated with glucose flux in human skeletal muscle. Diabetes August 1999 48:1508-1514; doi:10.2337/diabetes.48.8.1508
Relationship between insulin resistance and nonmodulating hypertension: linkage of metabolic abnormalities and cardiovascular
risk. Diabetes August 1999 48:1623-1630; doi:10.2337/diabetes.48.8.1623
Cellular immune response to phogrin in the NOD mouse: cloned T-cells cause destruction of islet transplants. Diabetes August 1999 48:1529-1534; doi:10.2337/diabetes.48.8.1529
Hyperinsulinism caused by paternal-specific inheritance of a recessive mutation in the sulfonylurea-receptor gene. Diabetes August 1999 48:1652-1657; doi:10.2337/diabetes.48.8.1652
Insulin-regulated mitochondrial gene expression is associated with glucose flux in human skeletal muscle. Diabetes August 1999 48:1508-1514; doi:10.2337/diabetes.48.8.1508
Insulin-regulated mitochondrial gene expression is associated with glucose flux in human skeletal muscle. Diabetes August 1999 48:1508-1514; doi:10.2337/diabetes.48.8.1508
Mechanism responsible for inactivation of skeletal muscle pyruvate dehydrogenase complex in starvation and diabetes. Diabetes August 1999 48:1593-1599; doi:10.2337/diabetes.48.8.1593
Augmentation of Ca2+-stimulated insulin release by glucose and long-chain fatty acids in rat pancreatic islets: free fatty
acids mimic ATP-sensitive K+ channel-independent insulinotropic action of glucose. Diabetes August 1999 48:1543-1549; doi:10.2337/diabetes.48.8.1543
Deficiency of phosphofructo-1-kinase/muscle subtype in humans is associated with impairment of insulin secretory oscillations.
Diabetes August 1999 48:1557-1561; doi:10.2337/diabetes.48.8.1557
Relationship between insulin resistance and nonmodulating hypertension: linkage of metabolic abnormalities and cardiovascular
risk. Diabetes August 1999 48:1623-1630; doi:10.2337/diabetes.48.8.1623
Glucose metabolism and insulin sensitivity in transgenic mice overexpressing leptin with lethal yellow agouti mutation: usefulness
of leptin for the treatment of obesity-associated diabetes. Diabetes August 1999 48:1615-1622; doi:10.2337/diabetes.48.8.1615
Insulin-regulated mitochondrial gene expression is associated with glucose flux in human skeletal muscle. Diabetes August 1999 48:1508-1514; doi:10.2337/diabetes.48.8.1508
Cellular immune response to phogrin in the NOD mouse: cloned T-cells cause destruction of islet transplants. Diabetes August 1999 48:1529-1534; doi:10.2337/diabetes.48.8.1529
Glucose metabolism and insulin sensitivity in transgenic mice overexpressing leptin with lethal yellow agouti mutation: usefulness
of leptin for the treatment of obesity-associated diabetes. Diabetes August 1999 48:1615-1622; doi:10.2337/diabetes.48.8.1615
Mechanism responsible for inactivation of skeletal muscle pyruvate dehydrogenase complex in starvation and diabetes. Diabetes August 1999 48:1593-1599; doi:10.2337/diabetes.48.8.1593
Glucose metabolism and insulin sensitivity in transgenic mice overexpressing leptin with lethal yellow agouti mutation: usefulness
of leptin for the treatment of obesity-associated diabetes. Diabetes August 1999 48:1615-1622; doi:10.2337/diabetes.48.8.1615
Glucose turnover and adipose tissue lipolysis are insulin-resistant in healthy relatives of type 2 diabetes patients: is cellular
insulin resistance a secondary phenomenon? Diabetes August 1999 48:1572-1578; doi:10.2337/diabetes.48.8.1572
Activation of the hexosamine pathway by glucosamine in vivo induces insulin resistance of early postreceptor insulin signaling
events in skeletal muscle. Diabetes August 1999 48:1562-1571; doi:10.2337/diabetes.48.8.1562
Augmentation of Ca2+-stimulated insulin release by glucose and long-chain fatty acids in rat pancreatic islets: free fatty
acids mimic ATP-sensitive K+ channel-independent insulinotropic action of glucose. Diabetes August 1999 48:1543-1549; doi:10.2337/diabetes.48.8.1543
Dehydroepiandrosterone suppresses the elevated hepatic glucose-6-phosphatase and fructose-1,6-bisphosphatase activities in
C57BL/Ksj-db/db mice: comparison with troglitazone. Diabetes August 1999 48:1579-1585; doi:10.2337/diabetes.48.8.1579
Insulin-regulated mitochondrial gene expression is associated with glucose flux in human skeletal muscle. Diabetes August 1999 48:1508-1514; doi:10.2337/diabetes.48.8.1508
Characterization of glucokinase mutations associated with maturity-onset diabetes of the young type 2 (MODY-2): different
glucokinase defects lead to a common phenotype. Diabetes August 1999 48:1645-1651; doi:10.2337/diabetes.48.8.1645
Dehydroepiandrosterone suppresses the elevated hepatic glucose-6-phosphatase and fructose-1,6-bisphosphatase activities in
C57BL/Ksj-db/db mice: comparison with troglitazone. Diabetes August 1999 48:1579-1585; doi:10.2337/diabetes.48.8.1579
Cellular immune response to phogrin in the NOD mouse: cloned T-cells cause destruction of islet transplants. Diabetes August 1999 48:1529-1534; doi:10.2337/diabetes.48.8.1529
Augmentation of Ca2+-stimulated insulin release by glucose and long-chain fatty acids in rat pancreatic islets: free fatty
acids mimic ATP-sensitive K+ channel-independent insulinotropic action of glucose. Diabetes August 1999 48:1543-1549; doi:10.2337/diabetes.48.8.1543
Insulin-regulated mitochondrial gene expression is associated with glucose flux in human skeletal muscle. Diabetes August 1999 48:1508-1514; doi:10.2337/diabetes.48.8.1508
Deficiency of phosphofructo-1-kinase/muscle subtype in humans is associated with impairment of insulin secretory oscillations.
Diabetes August 1999 48:1557-1561; doi:10.2337/diabetes.48.8.1557
Insulin-regulated mitochondrial gene expression is associated with glucose flux in human skeletal muscle. Diabetes August 1999 48:1508-1514; doi:10.2337/diabetes.48.8.1508
Activation of the hexosamine pathway by glucosamine in vivo induces insulin resistance of early postreceptor insulin signaling
events in skeletal muscle. Diabetes August 1999 48:1562-1571; doi:10.2337/diabetes.48.8.1562
Hyperinsulinism caused by paternal-specific inheritance of a recessive mutation in the sulfonylurea-receptor gene. Diabetes August 1999 48:1652-1657; doi:10.2337/diabetes.48.8.1652
Characterization of glucokinase mutations associated with maturity-onset diabetes of the young type 2 (MODY-2): different
glucokinase defects lead to a common phenotype. Diabetes August 1999 48:1645-1651; doi:10.2337/diabetes.48.8.1645
Characterization of glucokinase mutations associated with maturity-onset diabetes of the young type 2 (MODY-2): different
glucokinase defects lead to a common phenotype. Diabetes August 1999 48:1645-1651; doi:10.2337/diabetes.48.8.1645
Insulin-regulated mitochondrial gene expression is associated with glucose flux in human skeletal muscle. Diabetes August 1999 48:1508-1514; doi:10.2337/diabetes.48.8.1508
Insulin-regulated mitochondrial gene expression is associated with glucose flux in human skeletal muscle. Diabetes August 1999 48:1508-1514; doi:10.2337/diabetes.48.8.1508
Intramyocellular triglyceride content is a determinant of in vivo insulin resistance in humans: a 1H-13C nuclear magnetic
resonance spectroscopy assessment in offspring of type 2 diabetic parents. Diabetes August 1999 48:1600-1606; doi:10.2337/diabetes.48.8.1600
Glucose metabolism and insulin sensitivity in transgenic mice overexpressing leptin with lethal yellow agouti mutation: usefulness
of leptin for the treatment of obesity-associated diabetes. Diabetes August 1999 48:1615-1622; doi:10.2337/diabetes.48.8.1615
Characterization of glucokinase mutations associated with maturity-onset diabetes of the young type 2 (MODY-2): different
glucokinase defects lead to a common phenotype. Diabetes August 1999 48:1645-1651; doi:10.2337/diabetes.48.8.1645
Dehydroepiandrosterone suppresses the elevated hepatic glucose-6-phosphatase and fructose-1,6-bisphosphatase activities in
C57BL/Ksj-db/db mice: comparison with troglitazone. Diabetes August 1999 48:1579-1585; doi:10.2337/diabetes.48.8.1579
Deficiency of phosphofructo-1-kinase/muscle subtype in humans is associated with impairment of insulin secretory oscillations.
Diabetes August 1999 48:1557-1561; doi:10.2337/diabetes.48.8.1557
Glucose metabolism and insulin sensitivity in transgenic mice overexpressing leptin with lethal yellow agouti mutation: usefulness
of leptin for the treatment of obesity-associated diabetes. Diabetes August 1999 48:1615-1622; doi:10.2337/diabetes.48.8.1615
Glucose metabolism and insulin sensitivity in transgenic mice overexpressing leptin with lethal yellow agouti mutation: usefulness
of leptin for the treatment of obesity-associated diabetes. Diabetes August 1999 48:1615-1622; doi:10.2337/diabetes.48.8.1615
Glucose metabolism and insulin sensitivity in transgenic mice overexpressing leptin with lethal yellow agouti mutation: usefulness
of leptin for the treatment of obesity-associated diabetes. Diabetes August 1999 48:1615-1622; doi:10.2337/diabetes.48.8.1615
Dehydroepiandrosterone suppresses the elevated hepatic glucose-6-phosphatase and fructose-1,6-bisphosphatase activities in
C57BL/Ksj-db/db mice: comparison with troglitazone. Diabetes August 1999 48:1579-1585; doi:10.2337/diabetes.48.8.1579
Insulin-regulated mitochondrial gene expression is associated with glucose flux in human skeletal muscle. Diabetes August 1999 48:1508-1514; doi:10.2337/diabetes.48.8.1508
Deficiency of phosphofructo-1-kinase/muscle subtype in humans is associated with impairment of insulin secretory oscillations.
Diabetes August 1999 48:1557-1561; doi:10.2337/diabetes.48.8.1557
Intramyocellular triglyceride content is a determinant of in vivo insulin resistance in humans: a 1H-13C nuclear magnetic
resonance spectroscopy assessment in offspring of type 2 diabetic parents. Diabetes August 1999 48:1600-1606; doi:10.2337/diabetes.48.8.1600
Activation of the hexosamine pathway by glucosamine in vivo induces insulin resistance of early postreceptor insulin signaling
events in skeletal muscle. Diabetes August 1999 48:1562-1571; doi:10.2337/diabetes.48.8.1562
Intramyocellular triglyceride content is a determinant of in vivo insulin resistance in humans: a 1H-13C nuclear magnetic
resonance spectroscopy assessment in offspring of type 2 diabetic parents. Diabetes August 1999 48:1600-1606; doi:10.2337/diabetes.48.8.1600
Deficiency of phosphofructo-1-kinase/muscle subtype in humans is associated with impairment of insulin secretory oscillations.
Diabetes August 1999 48:1557-1561; doi:10.2337/diabetes.48.8.1557
Mechanism responsible for inactivation of skeletal muscle pyruvate dehydrogenase complex in starvation and diabetes. Diabetes August 1999 48:1593-1599; doi:10.2337/diabetes.48.8.1593
Intramyocellular triglyceride content is a determinant of in vivo insulin resistance in humans: a 1H-13C nuclear magnetic
resonance spectroscopy assessment in offspring of type 2 diabetic parents. Diabetes August 1999 48:1600-1606; doi:10.2337/diabetes.48.8.1600
Metabolic factors contributing to increased resting metabolic rate and decreased insulin-induced thermogenesis during the
development of type 2 diabetes. Diabetes August 1999 48:1607-1614; doi:10.2337/diabetes.48.8.1607
Plasma phospholipid transfer protein activity is lowered by 24-h insulin and acipimox administration: blunted response to
insulin in type 2 diabetic patients. Diabetes August 1999 48:1631-1637; doi:10.2337/diabetes.48.8.1631
Deficiency of phosphofructo-1-kinase/muscle subtype in humans is associated with impairment of insulin secretory oscillations.
Diabetes August 1999 48:1557-1561; doi:10.2337/diabetes.48.8.1557
Hyperinsulinism caused by paternal-specific inheritance of a recessive mutation in the sulfonylurea-receptor gene. Diabetes August 1999 48:1652-1657; doi:10.2337/diabetes.48.8.1652
Dehydroepiandrosterone suppresses the elevated hepatic glucose-6-phosphatase and fructose-1,6-bisphosphatase activities in
C57BL/Ksj-db/db mice: comparison with troglitazone. Diabetes August 1999 48:1579-1585; doi:10.2337/diabetes.48.8.1579
Relationship between insulin resistance and nonmodulating hypertension: linkage of metabolic abnormalities and cardiovascular
risk. Diabetes August 1999 48:1623-1630; doi:10.2337/diabetes.48.8.1623
Glucose metabolism and insulin sensitivity in transgenic mice overexpressing leptin with lethal yellow agouti mutation: usefulness
of leptin for the treatment of obesity-associated diabetes. Diabetes August 1999 48:1615-1622; doi:10.2337/diabetes.48.8.1615
Dehydroepiandrosterone suppresses the elevated hepatic glucose-6-phosphatase and fructose-1,6-bisphosphatase activities in
C57BL/Ksj-db/db mice: comparison with troglitazone. Diabetes August 1999 48:1579-1585; doi:10.2337/diabetes.48.8.1579
Augmentation of Ca2+-stimulated insulin release by glucose and long-chain fatty acids in rat pancreatic islets: free fatty
acids mimic ATP-sensitive K+ channel-independent insulinotropic action of glucose. Diabetes August 1999 48:1543-1549; doi:10.2337/diabetes.48.8.1543
Intramyocellular triglyceride content is a determinant of in vivo insulin resistance in humans: a 1H-13C nuclear magnetic
resonance spectroscopy assessment in offspring of type 2 diabetic parents. Diabetes August 1999 48:1600-1606; doi:10.2337/diabetes.48.8.1600
Dehydroepiandrosterone suppresses the elevated hepatic glucose-6-phosphatase and fructose-1,6-bisphosphatase activities in
C57BL/Ksj-db/db mice: comparison with troglitazone. Diabetes August 1999 48:1579-1585; doi:10.2337/diabetes.48.8.1579
Plasma phospholipid transfer protein activity is lowered by 24-h insulin and acipimox administration: blunted response to
insulin in type 2 diabetic patients. Diabetes August 1999 48:1631-1637; doi:10.2337/diabetes.48.8.1631
Glucose turnover and adipose tissue lipolysis are insulin-resistant in healthy relatives of type 2 diabetes patients: is cellular
insulin resistance a secondary phenomenon? Diabetes August 1999 48:1572-1578; doi:10.2337/diabetes.48.8.1572
Hyperinsulinism caused by paternal-specific inheritance of a recessive mutation in the sulfonylurea-receptor gene. Diabetes August 1999 48:1652-1657; doi:10.2337/diabetes.48.8.1652
Glucose metabolism and insulin sensitivity in transgenic mice overexpressing leptin with lethal yellow agouti mutation: usefulness
of leptin for the treatment of obesity-associated diabetes. Diabetes August 1999 48:1615-1622; doi:10.2337/diabetes.48.8.1615
Intramyocellular triglyceride content is a determinant of in vivo insulin resistance in humans: a 1H-13C nuclear magnetic
resonance spectroscopy assessment in offspring of type 2 diabetic parents. Diabetes August 1999 48:1600-1606; doi:10.2337/diabetes.48.8.1600
Hyperinsulinism caused by paternal-specific inheritance of a recessive mutation in the sulfonylurea-receptor gene. Diabetes August 1999 48:1652-1657; doi:10.2337/diabetes.48.8.1652
Insulin-regulated mitochondrial gene expression is associated with glucose flux in human skeletal muscle. Diabetes August 1999 48:1508-1514; doi:10.2337/diabetes.48.8.1508
Relationship between insulin resistance and nonmodulating hypertension: linkage of metabolic abnormalities and cardiovascular
risk. Diabetes August 1999 48:1623-1630; doi:10.2337/diabetes.48.8.1623
Plasma phospholipid transfer protein activity is lowered by 24-h insulin and acipimox administration: blunted response to
insulin in type 2 diabetic patients. Diabetes August 1999 48:1631-1637; doi:10.2337/diabetes.48.8.1631
Intramyocellular triglyceride content is a determinant of in vivo insulin resistance in humans: a 1H-13C nuclear magnetic
resonance spectroscopy assessment in offspring of type 2 diabetic parents. Diabetes August 1999 48:1600-1606; doi:10.2337/diabetes.48.8.1600
Activation of the hexosamine pathway by glucosamine in vivo induces insulin resistance of early postreceptor insulin signaling
events in skeletal muscle. Diabetes August 1999 48:1562-1571; doi:10.2337/diabetes.48.8.1562
Deficiency of phosphofructo-1-kinase/muscle subtype in humans is associated with impairment of insulin secretory oscillations.
Diabetes August 1999 48:1557-1561; doi:10.2337/diabetes.48.8.1557
Glucose turnover and adipose tissue lipolysis are insulin-resistant in healthy relatives of type 2 diabetes patients: is cellular
insulin resistance a secondary phenomenon? Diabetes August 1999 48:1572-1578; doi:10.2337/diabetes.48.8.1572
Cellular immune response to phogrin in the NOD mouse: cloned T-cells cause destruction of islet transplants. Diabetes August 1999 48:1529-1534; doi:10.2337/diabetes.48.8.1529
Metabolic factors contributing to increased resting metabolic rate and decreased insulin-induced thermogenesis during the
development of type 2 diabetes. Diabetes August 1999 48:1607-1614; doi:10.2337/diabetes.48.8.1607
Relationship between insulin resistance and nonmodulating hypertension: linkage of metabolic abnormalities and cardiovascular
risk. Diabetes August 1999 48:1623-1630; doi:10.2337/diabetes.48.8.1623
Mechanism responsible for inactivation of skeletal muscle pyruvate dehydrogenase complex in starvation and diabetes. Diabetes August 1999 48:1593-1599; doi:10.2337/diabetes.48.8.1593
Glucose turnover and adipose tissue lipolysis are insulin-resistant in healthy relatives of type 2 diabetes patients: is cellular
insulin resistance a secondary phenomenon? Diabetes August 1999 48:1572-1578; doi:10.2337/diabetes.48.8.1572
Augmentation of Ca2+-stimulated insulin release by glucose and long-chain fatty acids in rat pancreatic islets: free fatty
acids mimic ATP-sensitive K+ channel-independent insulinotropic action of glucose. Diabetes August 1999 48:1543-1549; doi:10.2337/diabetes.48.8.1543
Augmentation of Ca2+-stimulated insulin release by glucose and long-chain fatty acids in rat pancreatic islets: free fatty
acids mimic ATP-sensitive K+ channel-independent insulinotropic action of glucose. Diabetes August 1999 48:1543-1549; doi:10.2337/diabetes.48.8.1543
Augmentation of Ca2+-stimulated insulin release by glucose and long-chain fatty acids in rat pancreatic islets: free fatty
acids mimic ATP-sensitive K+ channel-independent insulinotropic action of glucose. Diabetes August 1999 48:1543-1549; doi:10.2337/diabetes.48.8.1543
Activation of the hexosamine pathway by glucosamine in vivo induces insulin resistance of early postreceptor insulin signaling
events in skeletal muscle. Diabetes August 1999 48:1562-1571; doi:10.2337/diabetes.48.8.1562
Glucose metabolism and insulin sensitivity in transgenic mice overexpressing leptin with lethal yellow agouti mutation: usefulness
of leptin for the treatment of obesity-associated diabetes. Diabetes August 1999 48:1615-1622; doi:10.2337/diabetes.48.8.1615