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Index by Author

November 2000; 49 (11)
  1. A
  2. B
  3. C
  4. D
  5. E
  6. F
  7. G
  8. H
  9. I
  10. J
  11. K
  12. L
  13. M
  14. N
  15. O
  16. P
  17. Q
  18. R
  19. S
  20. T
  21. U
  22. V
  23. W
  24. X
  25. Y
  26. Z

A

  1. Aizawa, S

    • Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia. Diabetes November 2000 49:1880-1889; doi:10.2337/diabetes.49.11.1880

  2. Akanuma, Y

    • Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia. Diabetes November 2000 49:1880-1889; doi:10.2337/diabetes.49.11.1880

  3. Andersen, H U

    • Cytokine- or chemically derived nitric oxide alters the expression of proteins detected by two-dimensional gel electrophoresis in neonatal rat islets of Langerhans. Diabetes November 2000 49:1819-1829; doi:10.2337/diabetes.49.11.1819

  4. Aoki, T

    • High glucose level and free fatty acid stimulate reactive oxygen species production through protein kinase C--dependent activation of NAD(P)H oxidase in cultured vascular cells. Diabetes November 2000 49:1939-1945; doi:10.2337/diabetes.49.11.1939

  5. Aragno, M

    • Dehydroepiandrosterone prevents oxidative injury induced by transient ischemia/reperfusion in the brain of diabetic rats. Diabetes November 2000 49:1924-1931; doi:10.2337/diabetes.49.11.1924

  6. Attie, A D

    • Genetic obesity unmasks nonlinear interactions between murine type 2 diabetes susceptibility loci. Diabetes November 2000 49:1946-1954; doi:10.2337/diabetes.49.11.1946

  7. Augert, G

    • Beacon: a novel gene involved in the regulation of energy balance. Diabetes November 2000 49:1766-1771; doi:10.2337/diabetes.49.11.1766

  8. Austin, D A

    • Stimulation of MAPK cascades by insulin and osmotic shock: lack of an involvement of p38 mitogen-activated protein kinase in glucose transport in 3T3-L1 adipocytes. Diabetes November 2000 49:1783-1793; doi:10.2337/diabetes.49.11.1783

B

  1. Beasley, J

    • Adrenalectomy reverses obese phenotype and restores hypothalamic melanocortin tone in leptin-deficient ob/ob mice. Diabetes November 2000 49:1917-1923; doi:10.2337/diabetes.49.11.1917

  2. Bell, G I

    • beta-cell transcription factors and diabetes: no evidence for diabetes-associated mutations in the gene encoding the basic helix-loop-helix transcription factor neurogenic differentiation 4 (NEUROD4) in Japanese patients with MODY. Diabetes November 2000 49:1955-1957; doi:10.2337/diabetes.49.11.1955

  3. Björklund, A

    • Glucose-induced [Ca2+]i abnormalities in human pancreatic islets: important role of overstimulation. Diabetes November 2000 49:1840-1848; doi:10.2337/diabetes.49.11.1840

  4. Bjørbaek, C

    • Unlike leptin, ciliary neurotrophic factor does not reverse the starvation-induced changes of serum corticosterone and hypothalamic neuropeptide levels but induces expression of hypothalamic inhibitors of leptin signaling. Diabetes November 2000 49:1890-1896; doi:10.2337/diabetes.49.11.1890

  5. Boccuzzi, G

    • Dehydroepiandrosterone prevents oxidative injury induced by transient ischemia/reperfusion in the brain of diabetic rats. Diabetes November 2000 49:1924-1931; doi:10.2337/diabetes.49.11.1924

  6. Bourut, C

    • Lamin A/C gene: sex-determined expression of mutations in Dunnigan-type familial partial lipodystrophy and absence of coding mutations in congenital and acquired generalized lipoatrophy. Diabetes November 2000 49:1958-1962; doi:10.2337/diabetes.49.11.1958

  7. Brignardello, E

    • Dehydroepiandrosterone prevents oxidative injury induced by transient ischemia/reperfusion in the brain of diabetic rats. Diabetes November 2000 49:1924-1931; doi:10.2337/diabetes.49.11.1924

C

  1. Capeau, J

    • Lamin A/C gene: sex-determined expression of mutations in Dunnigan-type familial partial lipodystrophy and absence of coding mutations in congenital and acquired generalized lipoatrophy. Diabetes November 2000 49:1958-1962; doi:10.2337/diabetes.49.11.1958

  2. Chin, G A

    • Impaired insulin secretion and increased insulin sensitivity in familial maturity-onset diabetes of the young 4 (insulin promoter factor 1 gene). Diabetes November 2000 49:1856-1864; doi:10.2337/diabetes.49.11.1856

  3. Christen, U

    • Virus-induced autoimmune diabetes: most beta-cells die through inflammatory cytokines and not perforin from autoreactive (anti-viral) cytotoxic T-lymphocytes. Diabetes November 2000 49:1801-1809; doi:10.2337/diabetes.49.11.1801

  4. Civitarese, A

    • Beacon: a novel gene involved in the regulation of energy balance. Diabetes November 2000 49:1766-1771; doi:10.2337/diabetes.49.11.1766

  5. Clarke, W L

    • Impaired insulin secretion and increased insulin sensitivity in familial maturity-onset diabetes of the young 4 (insulin promoter factor 1 gene). Diabetes November 2000 49:1856-1864; doi:10.2337/diabetes.49.11.1856

  6. Clark, M G

    • Acute impairment of insulin-mediated capillary recruitment and glucose uptake in rat skeletal muscle in vivo by TNF-alpha. Diabetes November 2000 49:1904-1909; doi:10.2337/diabetes.49.11.1904

  7. Clocquet, A R

    • Impaired insulin secretion and increased insulin sensitivity in familial maturity-onset diabetes of the young 4 (insulin promoter factor 1 gene). Diabetes November 2000 49:1856-1864; doi:10.2337/diabetes.49.11.1856

  8. Collier, G R

    • Beacon: a novel gene involved in the regulation of energy balance. Diabetes November 2000 49:1766-1771; doi:10.2337/diabetes.49.11.1766

  9. Coon, B

    • Virus-induced autoimmune diabetes: most beta-cells die through inflammatory cytokines and not perforin from autoreactive (anti-viral) cytotoxic T-lymphocytes. Diabetes November 2000 49:1801-1809; doi:10.2337/diabetes.49.11.1801

  10. Cooney, G J

    • Acyl-CoA inhibition of hexokinase in rat and human skeletal muscle is a potential mechanism of lipid-induced insulin resistance. Diabetes November 2000 49:1761-1765; doi:10.2337/diabetes.49.11.1761

  11. Costa, F

    • Effects of differing durations of antecedent hypoglycemia on counterregulatory responses to subsequent hypoglycemia in normal humans. Diabetes November 2000 49:1897-1903; doi:10.2337/diabetes.49.11.1897

  12. Côte, C H

    • Activation of p38 mitogen-activated protein kinase alpha and beta by insulin and contraction in rat skeletal muscle: potential role in the stimulation of glucose transport. Diabetes November 2000 49:1794-1800; doi:10.2337/diabetes.49.11.1794

  13. Cox, N J

    • beta-cell transcription factors and diabetes: no evidence for diabetes-associated mutations in the gene encoding the basic helix-loop-helix transcription factor neurogenic differentiation 4 (NEUROD4) in Japanese patients with MODY. Diabetes November 2000 49:1955-1957; doi:10.2337/diabetes.49.11.1955

D

  1. Danni, O

    • Dehydroepiandrosterone prevents oxidative injury induced by transient ischemia/reperfusion in the brain of diabetic rats. Diabetes November 2000 49:1924-1931; doi:10.2337/diabetes.49.11.1924

  2. Davis, S N

    • Effects of differing durations of antecedent hypoglycemia on counterregulatory responses to subsequent hypoglycemia in normal humans. Diabetes November 2000 49:1897-1903; doi:10.2337/diabetes.49.11.1897

  3. de Silva, A

    • Beacon: a novel gene involved in the regulation of energy balance. Diabetes November 2000 49:1766-1771; doi:10.2337/diabetes.49.11.1766

  4. Dodt, C

    • The subcutaneous lipolytic response to regional neural stimulation is reduced in obese women. Diabetes November 2000 49:1875-1879; doi:10.2337/diabetes.49.11.1875

  5. Dombrowski, L

    • Sustained activation of insulin receptors internalized in GLUT4 vesicles of insulin-stimulated skeletal muscle. Diabetes November 2000 49:1772-1782; doi:10.2337/diabetes.49.11.1772

  6. Druzhyna, N

    • Poly(ADP-ribose) polymerase facilitates the repair of N-methylpurines in mitochondrial DNA. Diabetes November 2000 49:1849-1855; doi:10.2337/diabetes.49.11.1849

E

  1. Egan, J M

    • Impaired insulin secretion and increased insulin sensitivity in familial maturity-onset diabetes of the young 4 (insulin promoter factor 1 gene). Diabetes November 2000 49:1856-1864; doi:10.2337/diabetes.49.11.1856

  2. Ejrnaes, M

    • Virus-induced autoimmune diabetes: most beta-cells die through inflammatory cytokines and not perforin from autoreactive (anti-viral) cytotoxic T-lymphocytes. Diabetes November 2000 49:1801-1809; doi:10.2337/diabetes.49.11.1801

  3. Elahi, D

    • Impaired insulin secretion and increased insulin sensitivity in familial maturity-onset diabetes of the young 4 (insulin promoter factor 1 gene). Diabetes November 2000 49:1856-1864; doi:10.2337/diabetes.49.11.1856

  4. Elam, M

    • The subcutaneous lipolytic response to regional neural stimulation is reduced in obese women. Diabetes November 2000 49:1875-1879; doi:10.2337/diabetes.49.11.1875

  5. Erani, D M

    • Unlike leptin, ciliary neurotrophic factor does not reverse the starvation-induced changes of serum corticosterone and hypothalamic neuropeptide levels but induces expression of hypothalamic inhibitors of leptin signaling. Diabetes November 2000 49:1890-1896; doi:10.2337/diabetes.49.11.1890

  6. Ertl, A C

    • Effects of differing durations of antecedent hypoglycemia on counterregulatory responses to subsequent hypoglycemia in normal humans. Diabetes November 2000 49:1897-1903; doi:10.2337/diabetes.49.11.1897

  7. Etoh, T

    • High glucose level and free fatty acid stimulate reactive oxygen species production through protein kinase C--dependent activation of NAD(P)H oxidase in cultured vascular cells. Diabetes November 2000 49:1939-1945; doi:10.2337/diabetes.49.11.1939

  8. Eto, K

    • Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia. Diabetes November 2000 49:1880-1889; doi:10.2337/diabetes.49.11.1880

F

  1. Faure, R

    • Sustained activation of insulin receptors internalized in GLUT4 vesicles of insulin-stimulated skeletal muscle. Diabetes November 2000 49:1772-1782; doi:10.2337/diabetes.49.11.1772

  2. Fehm, H L

    • The subcutaneous lipolytic response to regional neural stimulation is reduced in obese women. Diabetes November 2000 49:1875-1879; doi:10.2337/diabetes.49.11.1875

  3. Fey, S J

    • Cytokine- or chemically derived nitric oxide alters the expression of proteins detected by two-dimensional gel electrophoresis in neonatal rat islets of Langerhans. Diabetes November 2000 49:1819-1829; doi:10.2337/diabetes.49.11.1819

G

  1. Galassetti, P

    • Effects of differing durations of antecedent hypoglycemia on counterregulatory responses to subsequent hypoglycemia in normal humans. Diabetes November 2000 49:1897-1903; doi:10.2337/diabetes.49.11.1897

  2. Gavrilova, O

    • Lack of responses to a beta3-adrenergic agonist in lipoatrophic A-ZIP/F-1 mice. Diabetes November 2000 49:1910-1916; doi:10.2337/diabetes.49.11.1910

  3. German, M S

    • beta-cell transcription factors and diabetes: no evidence for diabetes-associated mutations in the gene encoding the basic helix-loop-helix transcription factor neurogenic differentiation 4 (NEUROD4) in Japanese patients with MODY. Diabetes November 2000 49:1955-1957; doi:10.2337/diabetes.49.11.1955

  4. Gong, Q

    • PIP2 and ATP cooperatively prevent cytosolic Ca2+-induced modification of ATP-sensitive K+ channels in rat pancreatic beta-cells. Diabetes November 2000 49:1830-1839; doi:10.2337/diabetes.49.11.1830

  5. Green, I C

    • Cytokine- or chemically derived nitric oxide alters the expression of proteins detected by two-dimensional gel electrophoresis in neonatal rat islets of Langerhans. Diabetes November 2000 49:1819-1829; doi:10.2337/diabetes.49.11.1819

  6. Grill, V E

    • Glucose-induced [Ca2+]i abnormalities in human pancreatic islets: important role of overstimulation. Diabetes November 2000 49:1840-1848; doi:10.2337/diabetes.49.11.1840

  7. Grimaldi, A

    • Lamin A/C gene: sex-determined expression of mutations in Dunnigan-type familial partial lipodystrophy and absence of coding mutations in congenital and acquired generalized lipoatrophy. Diabetes November 2000 49:1958-1962; doi:10.2337/diabetes.49.11.1958

  8. Guerci, B

    • Lamin A/C gene: sex-determined expression of mutations in Dunnigan-type familial partial lipodystrophy and absence of coding mutations in congenital and acquired generalized lipoatrophy. Diabetes November 2000 49:1958-1962; doi:10.2337/diabetes.49.11.1958

H

  1. Habener, J F

    • Impaired insulin secretion and increased insulin sensitivity in familial maturity-onset diabetes of the young 4 (insulin promoter factor 1 gene). Diabetes November 2000 49:1856-1864; doi:10.2337/diabetes.49.11.1856

  2. Hanks, J B

    • Impaired insulin secretion and increased insulin sensitivity in familial maturity-onset diabetes of the young 4 (insulin promoter factor 1 gene). Diabetes November 2000 49:1856-1864; doi:10.2337/diabetes.49.11.1856

  3. Hashimoto, T

    • High glucose level and free fatty acid stimulate reactive oxygen species production through protein kinase C--dependent activation of NAD(P)H oxidase in cultured vascular cells. Diabetes November 2000 49:1939-1945; doi:10.2337/diabetes.49.11.1939

  4. Henquin, J C

    • Triggering and amplifying pathways of regulation of insulin secretion by glucose. Diabetes November 2000 49:1751-1760; doi:10.2337/diabetes.49.11.1751

  5. Hileman, S M

    • Unlike leptin, ciliary neurotrophic factor does not reverse the starvation-induced changes of serum corticosterone and hypothalamic neuropeptide levels but induces expression of hypothalamic inhibitors of leptin signaling. Diabetes November 2000 49:1890-1896; doi:10.2337/diabetes.49.11.1890

  6. Hooton, J

    • Testicular sertoli cells protect islet beta-cells from autoimmune destruction in NOD mice by a transforming growth factor-beta1-dependent mechanism. Diabetes November 2000 49:1810-1818; doi:10.2337/diabetes.49.11.1810

  7. Horikawa, Y

    • beta-cell transcription factors and diabetes: no evidence for diabetes-associated mutations in the gene encoding the basic helix-loop-helix transcription factor neurogenic differentiation 4 (NEUROD4) in Japanese patients with MODY. Diabetes November 2000 49:1955-1957; doi:10.2337/diabetes.49.11.1955

I

  1. Ichinari, K

    • PIP2 and ATP cooperatively prevent cytosolic Ca2+-induced modification of ATP-sensitive K+ channels in rat pancreatic beta-cells. Diabetes November 2000 49:1830-1839; doi:10.2337/diabetes.49.11.1830

  2. Imamura, M

    • High glucose level and free fatty acid stimulate reactive oxygen species production through protein kinase C--dependent activation of NAD(P)H oxidase in cultured vascular cells. Diabetes November 2000 49:1939-1945; doi:10.2337/diabetes.49.11.1939

  3. Inoguchi, T

    • High glucose level and free fatty acid stimulate reactive oxygen species production through protein kinase C--dependent activation of NAD(P)H oxidase in cultured vascular cells. Diabetes November 2000 49:1939-1945; doi:10.2337/diabetes.49.11.1939

  4. Iwamoto, Y

    • beta-cell transcription factors and diabetes: no evidence for diabetes-associated mutations in the gene encoding the basic helix-loop-helix transcription factor neurogenic differentiation 4 (NEUROD4) in Japanese patients with MODY. Diabetes November 2000 49:1955-1957; doi:10.2337/diabetes.49.11.1955

  5. Iwasaki, N

    • beta-cell transcription factors and diabetes: no evidence for diabetes-associated mutations in the gene encoding the basic helix-loop-helix transcription factor neurogenic differentiation 4 (NEUROD4) in Japanese patients with MODY. Diabetes November 2000 49:1955-1957; doi:10.2337/diabetes.49.11.1955

J

  1. John, N E

    • Cytokine- or chemically derived nitric oxide alters the expression of proteins detected by two-dimensional gel electrophoresis in neonatal rat islets of Langerhans. Diabetes November 2000 49:1819-1829; doi:10.2337/diabetes.49.11.1819

  2. Jones, S

    • Beacon: a novel gene involved in the regulation of energy balance. Diabetes November 2000 49:1766-1771; doi:10.2337/diabetes.49.11.1766

K

  1. Kadowaki, T

    • Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia. Diabetes November 2000 49:1880-1889; doi:10.2337/diabetes.49.11.1880

  2. Kakei, M

    • PIP2 and ATP cooperatively prevent cytosolic Ca2+-induced modification of ATP-sensitive K+ channels in rat pancreatic beta-cells. Diabetes November 2000 49:1830-1839; doi:10.2337/diabetes.49.11.1830

  3. Kakimoto, M

    • High glucose level and free fatty acid stimulate reactive oxygen species production through protein kinase C--dependent activation of NAD(P)H oxidase in cultured vascular cells. Diabetes November 2000 49:1939-1945; doi:10.2337/diabetes.49.11.1939

  4. Kapur, S

    • Activation of p38 mitogen-activated protein kinase alpha and beta by insulin and contraction in rat skeletal muscle: potential role in the stimulation of glucose transport. Diabetes November 2000 49:1794-1800; doi:10.2337/diabetes.49.11.1794

  5. Karlsen, A E

    • Cytokine- or chemically derived nitric oxide alters the expression of proteins detected by two-dimensional gel electrophoresis in neonatal rat islets of Langerhans. Diabetes November 2000 49:1819-1829; doi:10.2337/diabetes.49.11.1819

  6. Kayali, A G

    • Stimulation of MAPK cascades by insulin and osmotic shock: lack of an involvement of p38 mitogen-activated protein kinase in glucose transport in 3T3-L1 adipocytes. Diabetes November 2000 49:1783-1793; doi:10.2337/diabetes.49.11.1783

  7. Kay, T W

    • Virus-induced autoimmune diabetes: most beta-cells die through inflammatory cytokines and not perforin from autoreactive (anti-viral) cytotoxic T-lymphocytes. Diabetes November 2000 49:1801-1809; doi:10.2337/diabetes.49.11.1801

  8. Keen, J

    • Activation of p38 mitogen-activated protein kinase alpha and beta by insulin and contraction in rat skeletal muscle: potential role in the stimulation of glucose transport. Diabetes November 2000 49:1794-1800; doi:10.2337/diabetes.49.11.1794

  9. Kim, D Y

    • Activation of p38 mitogen-activated protein kinase alpha and beta by insulin and contraction in rat skeletal muscle: potential role in the stimulation of glucose transport. Diabetes November 2000 49:1794-1800; doi:10.2337/diabetes.49.11.1794

  10. Kimura, S

    • Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia. Diabetes November 2000 49:1880-1889; doi:10.2337/diabetes.49.11.1880

  11. Klip, A

    • Activation of p38 mitogen-activated protein kinase alpha and beta by insulin and contraction in rat skeletal muscle: potential role in the stimulation of glucose transport. Diabetes November 2000 49:1794-1800; doi:10.2337/diabetes.49.11.1794

  12. Komeda, K

    • Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia. Diabetes November 2000 49:1880-1889; doi:10.2337/diabetes.49.11.1880

  13. Korbutt, G S

    • Testicular sertoli cells protect islet beta-cells from autoimmune destruction in NOD mice by a transforming growth factor-beta1-dependent mechanism. Diabetes November 2000 49:1810-1818; doi:10.2337/diabetes.49.11.1810

  14. Koriyama, N

    • PIP2 and ATP cooperatively prevent cytosolic Ca2+-induced modification of ATP-sensitive K+ channels in rat pancreatic beta-cells. Diabetes November 2000 49:1830-1839; doi:10.2337/diabetes.49.11.1830

  15. Kubota, N

    • Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia. Diabetes November 2000 49:1880-1889; doi:10.2337/diabetes.49.11.1880

L

  1. Lansner, A

    • Glucose-induced [Ca2+]i abnormalities in human pancreatic islets: important role of overstimulation. Diabetes November 2000 49:1840-1848; doi:10.2337/diabetes.49.11.1840

  2. Larsen, M R

    • Cytokine- or chemically derived nitric oxide alters the expression of proteins detected by two-dimensional gel electrophoresis in neonatal rat islets of Langerhans. Diabetes November 2000 49:1819-1829; doi:10.2337/diabetes.49.11.1819

  3. Larsen, P M

    • Cytokine- or chemically derived nitric oxide alters the expression of proteins detected by two-dimensional gel electrophoresis in neonatal rat islets of Langerhans. Diabetes November 2000 49:1819-1829; doi:10.2337/diabetes.49.11.1819

  4. Lascols, O

    • Lamin A/C gene: sex-determined expression of mutations in Dunnigan-type familial partial lipodystrophy and absence of coding mutations in congenital and acquired generalized lipoatrophy. Diabetes November 2000 49:1958-1962; doi:10.2337/diabetes.49.11.1958

  5. LeDoux, S P

    • Poly(ADP-ribose) polymerase facilitates the repair of N-methylpurines in mitochondrial DNA. Diabetes November 2000 49:1849-1855; doi:10.2337/diabetes.49.11.1849

  6. Lee, S

    • Beacon: a novel gene involved in the regulation of energy balance. Diabetes November 2000 49:1766-1771; doi:10.2337/diabetes.49.11.1766

  7. Lesniak, M

    • Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia. Diabetes November 2000 49:1880-1889; doi:10.2337/diabetes.49.11.1880

  8. Li, P

    • High glucose level and free fatty acid stimulate reactive oxygen species production through protein kinase C--dependent activation of NAD(P)H oxidase in cultured vascular cells. Diabetes November 2000 49:1939-1945; doi:10.2337/diabetes.49.11.1939

  9. Lloyd, D J

    • Lamin A/C gene: sex-determined expression of mutations in Dunnigan-type familial partial lipodystrophy and absence of coding mutations in congenital and acquired generalized lipoatrophy. Diabetes November 2000 49:1958-1962; doi:10.2337/diabetes.49.11.1958

  10. Lönnroth, P

    • The subcutaneous lipolytic response to regional neural stimulation is reduced in obese women. Diabetes November 2000 49:1875-1879; doi:10.2337/diabetes.49.11.1875

M

  1. Magré, J

    • Lamin A/C gene: sex-determined expression of mutations in Dunnigan-type familial partial lipodystrophy and absence of coding mutations in congenital and acquired generalized lipoatrophy. Diabetes November 2000 49:1958-1962; doi:10.2337/diabetes.49.11.1958

  2. Makimura, H

    • Adrenalectomy reverses obese phenotype and restores hypothalamic melanocortin tone in leptin-deficient ob/ob mice. Diabetes November 2000 49:1917-1923; doi:10.2337/diabetes.49.11.1917

  3. Mandrup-Poulsen, T

    • Cytokine- or chemically derived nitric oxide alters the expression of proteins detected by two-dimensional gel electrophoresis in neonatal rat islets of Langerhans. Diabetes November 2000 49:1819-1829; doi:10.2337/diabetes.49.11.1819

  4. Mann, S

    • Effects of differing durations of antecedent hypoglycemia on counterregulatory responses to subsequent hypoglycemia in normal humans. Diabetes November 2000 49:1897-1903; doi:10.2337/diabetes.49.11.1897

  5. Manti, R

    • Dehydroepiandrosterone prevents oxidative injury induced by transient ischemia/reperfusion in the brain of diabetic rats. Diabetes November 2000 49:1924-1931; doi:10.2337/diabetes.49.11.1924

  6. Mantzoros, C S

    • Unlike leptin, ciliary neurotrophic factor does not reverse the starvation-induced changes of serum corticosterone and hypothalamic neuropeptide levels but induces expression of hypothalamic inhibitors of leptin signaling. Diabetes November 2000 49:1890-1896; doi:10.2337/diabetes.49.11.1890

  7. Marcus-Samuels, B

    • Lack of responses to a beta3-adrenergic agonist in lipoatrophic A-ZIP/F-1 mice. Diabetes November 2000 49:1910-1916; doi:10.2337/diabetes.49.11.1910

  8. Marette, A

    • Sustained activation of insulin receptors internalized in GLUT4 vesicles of insulin-stimulated skeletal muscle. Diabetes November 2000 49:1772-1782; doi:10.2337/diabetes.49.11.1772
    • Activation of p38 mitogen-activated protein kinase alpha and beta by insulin and contraction in rat skeletal muscle: potential role in the stimulation of glucose transport. Diabetes November 2000 49:1794-1800; doi:10.2337/diabetes.49.11.1794

  9. Mauro, A

    • Dehydroepiandrosterone prevents oxidative injury induced by transient ischemia/reperfusion in the brain of diabetic rats. Diabetes November 2000 49:1924-1931; doi:10.2337/diabetes.49.11.1924

  10. McMillan, J S

    • Beacon: a novel gene involved in the regulation of energy balance. Diabetes November 2000 49:1766-1771; doi:10.2337/diabetes.49.11.1766

  11. Metz, S A

    • Genetic obesity unmasks nonlinear interactions between murine type 2 diabetes susceptibility loci. Diabetes November 2000 49:1946-1954; doi:10.2337/diabetes.49.11.1946

  12. Michelsen, B

    • Virus-induced autoimmune diabetes: most beta-cells die through inflammatory cytokines and not perforin from autoreactive (anti-viral) cytotoxic T-lymphocytes. Diabetes November 2000 49:1801-1809; doi:10.2337/diabetes.49.11.1801

  13. Miki, H

    • Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia. Diabetes November 2000 49:1880-1889; doi:10.2337/diabetes.49.11.1880

  14. Mizuno, T M

    • Adrenalectomy reverses obese phenotype and restores hypothalamic melanocortin tone in leptin-deficient ob/ob mice. Diabetes November 2000 49:1917-1923; doi:10.2337/diabetes.49.11.1917

  15. Mobbs, C V

    • Adrenalectomy reverses obese phenotype and restores hypothalamic melanocortin tone in leptin-deficient ob/ob mice. Diabetes November 2000 49:1917-1923; doi:10.2337/diabetes.49.11.1917

  16. Morimitsu, S

    • PIP2 and ATP cooperatively prevent cytosolic Ca2+-induced modification of ATP-sensitive K+ channels in rat pancreatic beta-cells. Diabetes November 2000 49:1830-1839; doi:10.2337/diabetes.49.11.1830

  17. Morton, G J

    • Beacon: a novel gene involved in the regulation of energy balance. Diabetes November 2000 49:1766-1771; doi:10.2337/diabetes.49.11.1766

  18. Muller, D C

    • Impaired insulin secretion and increased insulin sensitivity in familial maturity-onset diabetes of the young 4 (insulin promoter factor 1 gene). Diabetes November 2000 49:1856-1864; doi:10.2337/diabetes.49.11.1856

N

  1. Nadler, S T

    • Genetic obesity unmasks nonlinear interactions between murine type 2 diabetes susceptibility loci. Diabetes November 2000 49:1946-1954; doi:10.2337/diabetes.49.11.1946

  2. Nagai, R

    • Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia. Diabetes November 2000 49:1880-1889; doi:10.2337/diabetes.49.11.1880

  3. Nakano, R

    • Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia. Diabetes November 2000 49:1880-1889; doi:10.2337/diabetes.49.11.1880

  4. Nakazaki, M

    • PIP2 and ATP cooperatively prevent cytosolic Ca2+-induced modification of ATP-sensitive K+ channels in rat pancreatic beta-cells. Diabetes November 2000 49:1830-1839; doi:10.2337/diabetes.49.11.1830

  5. Naruse, M

    • High glucose level and free fatty acid stimulate reactive oxygen species production through protein kinase C--dependent activation of NAD(P)H oxidase in cultured vascular cells. Diabetes November 2000 49:1939-1945; doi:10.2337/diabetes.49.11.1939

  6. Nawata, H

    • High glucose level and free fatty acid stimulate reactive oxygen species production through protein kinase C--dependent activation of NAD(P)H oxidase in cultured vascular cells. Diabetes November 2000 49:1939-1945; doi:10.2337/diabetes.49.11.1939

  7. Neill, R A

    • Effects of differing durations of antecedent hypoglycemia on counterregulatory responses to subsequent hypoglycemia in normal humans. Diabetes November 2000 49:1897-1903; doi:10.2337/diabetes.49.11.1897

  8. Nerup, J

    • Cytokine- or chemically derived nitric oxide alters the expression of proteins detected by two-dimensional gel electrophoresis in neonatal rat islets of Langerhans. Diabetes November 2000 49:1819-1829; doi:10.2337/diabetes.49.11.1819

O

  1. Ogata, M

    • beta-cell transcription factors and diabetes: no evidence for diabetes-associated mutations in the gene encoding the basic helix-loop-helix transcription factor neurogenic differentiation 4 (NEUROD4) in Japanese patients with MODY. Diabetes November 2000 49:1955-1957; doi:10.2337/diabetes.49.11.1955

P

  1. Padova, G

    • Lamin A/C gene: sex-determined expression of mutations in Dunnigan-type familial partial lipodystrophy and absence of coding mutations in congenital and acquired generalized lipoatrophy. Diabetes November 2000 49:1958-1962; doi:10.2337/diabetes.49.11.1958

  2. Parola, S

    • Dehydroepiandrosterone prevents oxidative injury induced by transient ischemia/reperfusion in the brain of diabetic rats. Diabetes November 2000 49:1924-1931; doi:10.2337/diabetes.49.11.1924

  3. Perreault, M

    • Activation of p38 mitogen-activated protein kinase alpha and beta by insulin and contraction in rat skeletal muscle: potential role in the stimulation of glucose transport. Diabetes November 2000 49:1794-1800; doi:10.2337/diabetes.49.11.1794

  4. Piquemal, R

    • Lamin A/C gene: sex-determined expression of mutations in Dunnigan-type familial partial lipodystrophy and absence of coding mutations in congenital and acquired generalized lipoatrophy. Diabetes November 2000 49:1958-1962; doi:10.2337/diabetes.49.11.1958

  5. Pociot, F

    • Cytokine- or chemically derived nitric oxide alters the expression of proteins detected by two-dimensional gel electrophoresis in neonatal rat islets of Langerhans. Diabetes November 2000 49:1819-1829; doi:10.2337/diabetes.49.11.1819

  6. Power, R

    • Testicular sertoli cells protect islet beta-cells from autoimmune destruction in NOD mice by a transforming growth factor-beta1-dependent mechanism. Diabetes November 2000 49:1810-1818; doi:10.2337/diabetes.49.11.1810

R

  1. Rabaglia, M E

    • Genetic obesity unmasks nonlinear interactions between murine type 2 diabetes susceptibility loci. Diabetes November 2000 49:1946-1954; doi:10.2337/diabetes.49.11.1946

  2. Rabinovitch, A

    • Testicular sertoli cells protect islet beta-cells from autoimmune destruction in NOD mice by a transforming growth factor-beta1-dependent mechanism. Diabetes November 2000 49:1810-1818; doi:10.2337/diabetes.49.11.1810

  3. Rajotte, R V

    • Testicular sertoli cells protect islet beta-cells from autoimmune destruction in NOD mice by a transforming growth factor-beta1-dependent mechanism. Diabetes November 2000 49:1810-1818; doi:10.2337/diabetes.49.11.1810

  4. Ramlal, T

    • Activation of p38 mitogen-activated protein kinase alpha and beta by insulin and contraction in rat skeletal muscle: potential role in the stimulation of glucose transport. Diabetes November 2000 49:1794-1800; doi:10.2337/diabetes.49.11.1794

  5. Rattigan, S

    • Acute impairment of insulin-mediated capillary recruitment and glucose uptake in rat skeletal muscle in vivo by TNF-alpha. Diabetes November 2000 49:1904-1909; doi:10.2337/diabetes.49.11.1904

  6. Reitman, M L

    • Lack of responses to a beta3-adrenergic agonist in lipoatrophic A-ZIP/F-1 mice. Diabetes November 2000 49:1910-1916; doi:10.2337/diabetes.49.11.1910

  7. Ritter, S

    • Repeated 2-deoxy-D-glucose-induced glucoprivation attenuates Fos expression and glucoregulatory responses during subsequent glucoprivation. Diabetes November 2000 49:1865-1874; doi:10.2337/diabetes.49.11.1865

  8. Roberts, J

    • Adrenalectomy reverses obese phenotype and restores hypothalamic melanocortin tone in leptin-deficient ob/ob mice. Diabetes November 2000 49:1917-1923; doi:10.2337/diabetes.49.11.1917

  9. Rodrigo, E

    • Virus-induced autoimmune diabetes: most beta-cells die through inflammatory cytokines and not perforin from autoreactive (anti-viral) cytotoxic T-lymphocytes. Diabetes November 2000 49:1801-1809; doi:10.2337/diabetes.49.11.1801

  10. Roepstorff, P

    • Cytokine- or chemically derived nitric oxide alters the expression of proteins detected by two-dimensional gel electrophoresis in neonatal rat islets of Langerhans. Diabetes November 2000 49:1819-1829; doi:10.2337/diabetes.49.11.1819

S

  1. Sanders, N M

    • Repeated 2-deoxy-D-glucose-induced glucoprivation attenuates Fos expression and glucoregulatory responses during subsequent glucoprivation. Diabetes November 2000 49:1865-1874; doi:10.2337/diabetes.49.11.1865

  2. Sano, H

    • High glucose level and free fatty acid stimulate reactive oxygen species production through protein kinase C--dependent activation of NAD(P)H oxidase in cultured vascular cells. Diabetes November 2000 49:1939-1945; doi:10.2337/diabetes.49.11.1939

  3. Satoh, S

    • Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia. Diabetes November 2000 49:1880-1889; doi:10.2337/diabetes.49.11.1880

  4. Schueler, K L

    • Genetic obesity unmasks nonlinear interactions between murine type 2 diabetes susceptibility loci. Diabetes November 2000 49:1946-1954; doi:10.2337/diabetes.49.11.1946

  5. Schwitzgebel, V

    • beta-cell transcription factors and diabetes: no evidence for diabetes-associated mutations in the gene encoding the basic helix-loop-helix transcription factor neurogenic differentiation 4 (NEUROD4) in Japanese patients with MODY. Diabetes November 2000 49:1955-1957; doi:10.2337/diabetes.49.11.1955

  6. Sciacchitano, S

    • Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia. Diabetes November 2000 49:1880-1889; doi:10.2337/diabetes.49.11.1880

  7. Seewaldt, S

    • Virus-induced autoimmune diabetes: most beta-cells die through inflammatory cytokines and not perforin from autoreactive (anti-viral) cytotoxic T-lymphocytes. Diabetes November 2000 49:1801-1809; doi:10.2337/diabetes.49.11.1801

  8. Sekihara, H

    • Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia. Diabetes November 2000 49:1880-1889; doi:10.2337/diabetes.49.11.1880

  9. Shackleton, S

    • Lamin A/C gene: sex-determined expression of mutations in Dunnigan-type familial partial lipodystrophy and absence of coding mutations in congenital and acquired generalized lipoatrophy. Diabetes November 2000 49:1958-1962; doi:10.2337/diabetes.49.11.1958

  10. Silverstein, J

    • Adrenalectomy reverses obese phenotype and restores hypothalamic melanocortin tone in leptin-deficient ob/ob mice. Diabetes November 2000 49:1917-1923; doi:10.2337/diabetes.49.11.1917

  11. Smulson, M E

    • Poly(ADP-ribose) polymerase facilitates the repair of N-methylpurines in mitochondrial DNA. Diabetes November 2000 49:1849-1855; doi:10.2337/diabetes.49.11.1849

  12. Somwar, R

    • Activation of p38 mitogen-activated protein kinase alpha and beta by insulin and contraction in rat skeletal muscle: potential role in the stimulation of glucose transport. Diabetes November 2000 49:1794-1800; doi:10.2337/diabetes.49.11.1794

  13. Srinivasan, S

    • Diabetic peripheral neuropathy: evidence for apoptosis and associated mitochondrial dysfunction. Diabetes November 2000 49:1932-1938; doi:10.2337/diabetes.49.11.1932

  14. Stevens, M

    • Diabetic peripheral neuropathy: evidence for apoptosis and associated mitochondrial dysfunction. Diabetes November 2000 49:1932-1938; doi:10.2337/diabetes.49.11.1932

  15. Stoehr, J P

    • Genetic obesity unmasks nonlinear interactions between murine type 2 diabetes susceptibility loci. Diabetes November 2000 49:1946-1954; doi:10.2337/diabetes.49.11.1946

  16. Stoffers, D A

    • Impaired insulin secretion and increased insulin sensitivity in familial maturity-onset diabetes of the young 4 (insulin promoter factor 1 gene). Diabetes November 2000 49:1856-1864; doi:10.2337/diabetes.49.11.1856

  17. Suarez-Pinzon, W

    • Testicular sertoli cells protect islet beta-cells from autoimmune destruction in NOD mice by a transforming growth factor-beta1-dependent mechanism. Diabetes November 2000 49:1810-1818; doi:10.2337/diabetes.49.11.1810

  18. Suzuki, R

    • Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia. Diabetes November 2000 49:1880-1889; doi:10.2337/diabetes.49.11.1880

  19. Sweeney, G

    • Activation of p38 mitogen-activated protein kinase alpha and beta by insulin and contraction in rat skeletal muscle: potential role in the stimulation of glucose transport. Diabetes November 2000 49:1794-1800; doi:10.2337/diabetes.49.11.1794

T

  1. Taha, C

    • Activation of p38 mitogen-activated protein kinase alpha and beta by insulin and contraction in rat skeletal muscle: potential role in the stimulation of glucose transport. Diabetes November 2000 49:1794-1800; doi:10.2337/diabetes.49.11.1794

  2. Tamagno, E

    • Dehydroepiandrosterone prevents oxidative injury induced by transient ischemia/reperfusion in the brain of diabetic rats. Diabetes November 2000 49:1924-1931; doi:10.2337/diabetes.49.11.1924

  3. Tate, D

    • Effects of differing durations of antecedent hypoglycemia on counterregulatory responses to subsequent hypoglycemia in normal humans. Diabetes November 2000 49:1897-1903; doi:10.2337/diabetes.49.11.1897

  4. Taylor, S I

    • Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia. Diabetes November 2000 49:1880-1889; doi:10.2337/diabetes.49.11.1880

  5. Tei, C

    • PIP2 and ATP cooperatively prevent cytosolic Ca2+-induced modification of ATP-sensitive K+ channels in rat pancreatic beta-cells. Diabetes November 2000 49:1830-1839; doi:10.2337/diabetes.49.11.1830

  6. Tenne-Brown, J

    • Beacon: a novel gene involved in the regulation of energy balance. Diabetes November 2000 49:1766-1771; doi:10.2337/diabetes.49.11.1766

  7. Terauchi, Y

    • Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia. Diabetes November 2000 49:1880-1889; doi:10.2337/diabetes.49.11.1880

  8. Thomas, H E

    • Virus-induced autoimmune diabetes: most beta-cells die through inflammatory cytokines and not perforin from autoreactive (anti-viral) cytotoxic T-lymphocytes. Diabetes November 2000 49:1801-1809; doi:10.2337/diabetes.49.11.1801

  9. Thompson, A L

    • Acyl-CoA inhibition of hexokinase in rat and human skeletal muscle is a potential mechanism of lipid-induced insulin resistance. Diabetes November 2000 49:1761-1765; doi:10.2337/diabetes.49.11.1761

  10. Tobe, K

    • Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia. Diabetes November 2000 49:1880-1889; doi:10.2337/diabetes.49.11.1880

  11. Touraine, P

    • Lamin A/C gene: sex-determined expression of mutations in Dunnigan-type familial partial lipodystrophy and absence of coding mutations in congenital and acquired generalized lipoatrophy. Diabetes November 2000 49:1958-1962; doi:10.2337/diabetes.49.11.1958

  12. Trembath, R C

    • Lamin A/C gene: sex-determined expression of mutations in Dunnigan-type familial partial lipodystrophy and absence of coding mutations in congenital and acquired generalized lipoatrophy. Diabetes November 2000 49:1958-1962; doi:10.2337/diabetes.49.11.1958

  13. Trevaskis, J

    • Beacon: a novel gene involved in the regulation of energy balance. Diabetes November 2000 49:1766-1771; doi:10.2337/diabetes.49.11.1766

  14. Tsubamoto, Y

    • Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia. Diabetes November 2000 49:1880-1889; doi:10.2337/diabetes.49.11.1880

U

  1. Umeda, F

    • High glucose level and free fatty acid stimulate reactive oxygen species production through protein kinase C--dependent activation of NAD(P)H oxidase in cultured vascular cells. Diabetes November 2000 49:1939-1945; doi:10.2337/diabetes.49.11.1939

  2. Utsumi, H

    • High glucose level and free fatty acid stimulate reactive oxygen species production through protein kinase C--dependent activation of NAD(P)H oxidase in cultured vascular cells. Diabetes November 2000 49:1939-1945; doi:10.2337/diabetes.49.11.1939

V

  1. Valensi, P

    • Lamin A/C gene: sex-determined expression of mutations in Dunnigan-type familial partial lipodystrophy and absence of coding mutations in congenital and acquired generalized lipoatrophy. Diabetes November 2000 49:1958-1962; doi:10.2337/diabetes.49.11.1958

  2. Vantyghem, M C

    • Lamin A/C gene: sex-determined expression of mutations in Dunnigan-type familial partial lipodystrophy and absence of coding mutations in congenital and acquired generalized lipoatrophy. Diabetes November 2000 49:1958-1962; doi:10.2337/diabetes.49.11.1958

  3. Vigouroux, C

    • Lamin A/C gene: sex-determined expression of mutations in Dunnigan-type familial partial lipodystrophy and absence of coding mutations in congenital and acquired generalized lipoatrophy. Diabetes November 2000 49:1958-1962; doi:10.2337/diabetes.49.11.1958

  4. von Herrath, M G

    • Virus-induced autoimmune diabetes: most beta-cells die through inflammatory cytokines and not perforin from autoreactive (anti-viral) cytotoxic T-lymphocytes. Diabetes November 2000 49:1801-1809; doi:10.2337/diabetes.49.11.1801

W

  1. Walder, K

    • Beacon: a novel gene involved in the regulation of energy balance. Diabetes November 2000 49:1766-1771; doi:10.2337/diabetes.49.11.1766

  2. Webster, N J

    • Stimulation of MAPK cascades by insulin and osmotic shock: lack of an involvement of p38 mitogen-activated protein kinase in glucose transport in 3T3-L1 adipocytes. Diabetes November 2000 49:1783-1793; doi:10.2337/diabetes.49.11.1783

  3. Wideman, L

    • Impaired insulin secretion and increased insulin sensitivity in familial maturity-onset diabetes of the young 4 (insulin promoter factor 1 gene). Diabetes November 2000 49:1856-1864; doi:10.2337/diabetes.49.11.1856

  4. Wiley, J W

    • Diabetic peripheral neuropathy: evidence for apoptosis and associated mitochondrial dysfunction. Diabetes November 2000 49:1932-1938; doi:10.2337/diabetes.49.11.1932

  5. Wilson, G L

    • Poly(ADP-ribose) polymerase facilitates the repair of N-methylpurines in mitochondrial DNA. Diabetes November 2000 49:1849-1855; doi:10.2337/diabetes.49.11.1849

  6. Windmill, K

    • Beacon: a novel gene involved in the regulation of energy balance. Diabetes November 2000 49:1766-1771; doi:10.2337/diabetes.49.11.1766

  7. Wolfe, T

    • Virus-induced autoimmune diabetes: most beta-cells die through inflammatory cytokines and not perforin from autoreactive (anti-viral) cytotoxic T-lymphocytes. Diabetes November 2000 49:1801-1809; doi:10.2337/diabetes.49.11.1801

Y

  1. Yada, T

    • PIP2 and ATP cooperatively prevent cytosolic Ca2+-induced modification of ATP-sensitive K+ channels in rat pancreatic beta-cells. Diabetes November 2000 49:1830-1839; doi:10.2337/diabetes.49.11.1830

  2. Yaekura, K

    • PIP2 and ATP cooperatively prevent cytosolic Ca2+-induced modification of ATP-sensitive K+ channels in rat pancreatic beta-cells. Diabetes November 2000 49:1830-1839; doi:10.2337/diabetes.49.11.1830

  3. Yamauchi, T

    • Disruption of insulin receptor substrate 2 causes type 2 diabetes because of liver insulin resistance and lack of compensatory beta-cell hyperplasia. Diabetes November 2000 49:1880-1889; doi:10.2337/diabetes.49.11.1880

  4. Yandell, B S

    • Genetic obesity unmasks nonlinear interactions between murine type 2 diabetes susceptibility loci. Diabetes November 2000 49:1946-1954; doi:10.2337/diabetes.49.11.1946

  5. Youd, J M

    • Acute impairment of insulin-mediated capillary recruitment and glucose uptake in rat skeletal muscle in vivo by TNF-alpha. Diabetes November 2000 49:1904-1909; doi:10.2337/diabetes.49.11.1904

  6. Yu, H Y

    • High glucose level and free fatty acid stimulate reactive oxygen species production through protein kinase C--dependent activation of NAD(P)H oxidase in cultured vascular cells. Diabetes November 2000 49:1939-1945; doi:10.2337/diabetes.49.11.1939

Z

  1. Zimmet, P Z

    • Beacon: a novel gene involved in the regulation of energy balance. Diabetes November 2000 49:1766-1771; doi:10.2337/diabetes.49.11.1766

  2. Ziotopoulou, M

    • Unlike leptin, ciliary neurotrophic factor does not reverse the starvation-induced changes of serum corticosterone and hypothalamic neuropeptide levels but induces expression of hypothalamic inhibitors of leptin signaling. Diabetes November 2000 49:1890-1896; doi:10.2337/diabetes.49.11.1890

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