Human Metabolic Syndrome Resulting From Dominant-Negative Mutations in the Nuclear Receptor Peroxisome Proliferator-Activated
Receptor-γ Diabetes April 2003 52:910-917; doi:10.2337/diabetes.52.4.910
Human Metabolic Syndrome Resulting From Dominant-Negative Mutations in the Nuclear Receptor Peroxisome Proliferator-Activated
Receptor-γ Diabetes April 2003 52:910-917; doi:10.2337/diabetes.52.4.910
Evaluation of Autofluorescent Property of Hemoglobin-Advanced Glycation End Product as a Long-Term Glycemic Index of Diabetes
Diabetes April 2003 52:1041-1046; doi:10.2337/diabetes.52.4.1041
Inhibition of Mitochondrial Na+-Ca2+ Exchanger Increases Mitochondrial Metabolism and Potentiates Glucose-Stimulated Insulin Secretion in Rat Pancreatic Islets
Diabetes April 2003 52:965-973; doi:10.2337/diabetes.52.4.965
Elevation in Tanis Expression Alters Glucose Metabolism and Insulin Sensitivity in H4IIE Cells Diabetes April 2003 52:929-934; doi:10.2337/diabetes.52.4.929
Human Metabolic Syndrome Resulting From Dominant-Negative Mutations in the Nuclear Receptor Peroxisome Proliferator-Activated
Receptor-γ Diabetes April 2003 52:910-917; doi:10.2337/diabetes.52.4.910
Elevation in Tanis Expression Alters Glucose Metabolism and Insulin Sensitivity in H4IIE Cells Diabetes April 2003 52:929-934; doi:10.2337/diabetes.52.4.929
Human Metabolic Syndrome Resulting From Dominant-Negative Mutations in the Nuclear Receptor Peroxisome Proliferator-Activated
Receptor-γ Diabetes April 2003 52:910-917; doi:10.2337/diabetes.52.4.910
Protein Kinase C δ Activation and Translocation to the Nucleus Are Required for Fatty Acid-Induced Apoptosis of Insulin-Secreting
Cells Diabetes April 2003 52:991-997; doi:10.2337/diabetes.52.4.991
Protein Kinase C δ Activation and Translocation to the Nucleus Are Required for Fatty Acid-Induced Apoptosis of Insulin-Secreting
Cells Diabetes April 2003 52:991-997; doi:10.2337/diabetes.52.4.991
Protein Kinase C δ Activation and Translocation to the Nucleus Are Required for Fatty Acid-Induced Apoptosis of Insulin-Secreting
Cells Diabetes April 2003 52:991-997; doi:10.2337/diabetes.52.4.991
Differential Activation Mechanisms of Erk-1/2 and p70S6K by Glucose in Pancreatic β-Cells
Diabetes April 2003 52:974-983; doi:10.2337/diabetes.52.4.974
Defective Signaling Through Akt-2 and -3 But Not Akt-1 in Insulin-Resistant Human Skeletal Muscle: Potential Role in Insulin Resistance Diabetes April 2003 52:935-941; doi:10.2337/diabetes.52.4.935
Nephrin Expression Is Reduced in Human Diabetic Nephropathy: Evidence for a Distinct Role for Glycated Albumin and Angiotensin II Diabetes April 2003 52:1023-1030; doi:10.2337/diabetes.52.4.1023
Low Glomerular Filtration Rate in Normoalbuminuric Type 1 Diabetic Patients: An Indicator of More Advanced Glomerular Lesions Diabetes April 2003 52:1036-1040; doi:10.2337/diabetes.52.4.1036
Human Metabolic Syndrome Resulting From Dominant-Negative Mutations in the Nuclear Receptor Peroxisome Proliferator-Activated
Receptor-γ Diabetes April 2003 52:910-917; doi:10.2337/diabetes.52.4.910
Human Metabolic Syndrome Resulting From Dominant-Negative Mutations in the Nuclear Receptor Peroxisome Proliferator-Activated
Receptor-γ Diabetes April 2003 52:910-917; doi:10.2337/diabetes.52.4.910
Elevation in Tanis Expression Alters Glucose Metabolism and Insulin Sensitivity in H4IIE Cells Diabetes April 2003 52:929-934; doi:10.2337/diabetes.52.4.929
Is Podocyte Injury Relevant in Diabetic Nephropathy?: Studies in Patients With Type 2 Diabetes Diabetes April 2003 52:1031-1035; doi:10.2337/diabetes.52.4.1031
Is Podocyte Injury Relevant in Diabetic Nephropathy?: Studies in Patients With Type 2 Diabetes Diabetes April 2003 52:1031-1035; doi:10.2337/diabetes.52.4.1031
Nephrin Expression Is Reduced in Human Diabetic Nephropathy: Evidence for a Distinct Role for Glycated Albumin and Angiotensin II Diabetes April 2003 52:1023-1030; doi:10.2337/diabetes.52.4.1023
Elevation in Tanis Expression Alters Glucose Metabolism and Insulin Sensitivity in H4IIE Cells Diabetes April 2003 52:929-934; doi:10.2337/diabetes.52.4.929
Differential Activation Mechanisms of Erk-1/2 and p70S6K by Glucose in Pancreatic β-Cells
Diabetes April 2003 52:974-983; doi:10.2337/diabetes.52.4.974
Human Metabolic Syndrome Resulting From Dominant-Negative Mutations in the Nuclear Receptor Peroxisome Proliferator-Activated
Receptor-γ Diabetes April 2003 52:910-917; doi:10.2337/diabetes.52.4.910
Defective Signaling Through Akt-2 and -3 But Not Akt-1 in Insulin-Resistant Human Skeletal Muscle: Potential Role in Insulin Resistance Diabetes April 2003 52:935-941; doi:10.2337/diabetes.52.4.935
Mechanisms for the Deterioration in Glucose Tolerance Associated With HIV Protease Inhibitor Regimens Diabetes April 2003 52:918-925; doi:10.2337/diabetes.52.4.918
Nephrin Expression Is Reduced in Human Diabetic Nephropathy: Evidence for a Distinct Role for Glycated Albumin and Angiotensin II Diabetes April 2003 52:1023-1030; doi:10.2337/diabetes.52.4.1023
Human Metabolic Syndrome Resulting From Dominant-Negative Mutations in the Nuclear Receptor Peroxisome Proliferator-Activated
Receptor-γ Diabetes April 2003 52:910-917; doi:10.2337/diabetes.52.4.910
Protein Kinase C δ Activation and Translocation to the Nucleus Are Required for Fatty Acid-Induced Apoptosis of Insulin-Secreting
Cells Diabetes April 2003 52:991-997; doi:10.2337/diabetes.52.4.991
Elevation in Tanis Expression Alters Glucose Metabolism and Insulin Sensitivity in H4IIE Cells Diabetes April 2003 52:929-934; doi:10.2337/diabetes.52.4.929
Is Podocyte Injury Relevant in Diabetic Nephropathy?: Studies in Patients With Type 2 Diabetes Diabetes April 2003 52:1031-1035; doi:10.2337/diabetes.52.4.1031
Low Glomerular Filtration Rate in Normoalbuminuric Type 1 Diabetic Patients: An Indicator of More Advanced Glomerular Lesions Diabetes April 2003 52:1036-1040; doi:10.2337/diabetes.52.4.1036
Ghrelin Directly Interacts With Neuropeptide-Y-Containing Neurons in the Rat Arcuate Nucleus: Ca2+ Signaling via Protein Kinase A and N-Type Channel-Dependent Mechanisms and Cross-Talk With Leptin and Orexin
Diabetes April 2003 52:948-956; doi:10.2337/diabetes.52.4.948
Elevation in Tanis Expression Alters Glucose Metabolism and Insulin Sensitivity in H4IIE Cells Diabetes April 2003 52:929-934; doi:10.2337/diabetes.52.4.929
Mechanisms for the Deterioration in Glucose Tolerance Associated With HIV Protease Inhibitor Regimens Diabetes April 2003 52:918-925; doi:10.2337/diabetes.52.4.918
Inhibition of Mitochondrial Na+-Ca2+ Exchanger Increases Mitochondrial Metabolism and Potentiates Glucose-Stimulated Insulin Secretion in Rat Pancreatic Islets
Diabetes April 2003 52:965-973; doi:10.2337/diabetes.52.4.965
Evaluation of Autofluorescent Property of Hemoglobin-Advanced Glycation End Product as a Long-Term Glycemic Index of Diabetes
Diabetes April 2003 52:1041-1046; doi:10.2337/diabetes.52.4.1041
Inhibition of Mitochondrial Na+-Ca2+ Exchanger Increases Mitochondrial Metabolism and Potentiates Glucose-Stimulated Insulin Secretion in Rat Pancreatic Islets
Diabetes April 2003 52:965-973; doi:10.2337/diabetes.52.4.965
Human Metabolic Syndrome Resulting From Dominant-Negative Mutations in the Nuclear Receptor Peroxisome Proliferator-Activated
Receptor-γ Diabetes April 2003 52:910-917; doi:10.2337/diabetes.52.4.910
Protein Kinase C δ Activation and Translocation to the Nucleus Are Required for Fatty Acid-Induced Apoptosis of Insulin-Secreting
Cells Diabetes April 2003 52:991-997; doi:10.2337/diabetes.52.4.991
Genetic Liability of Type 1 Diabetes and the Onset Age Among 22,650 Young Finnish Twin Pairs: A Nationwide Follow-Up Study Diabetes April 2003 52:1052-1055; doi:10.2337/diabetes.52.4.1052
Human Metabolic Syndrome Resulting From Dominant-Negative Mutations in the Nuclear Receptor Peroxisome Proliferator-Activated
Receptor-γ Diabetes April 2003 52:910-917; doi:10.2337/diabetes.52.4.910
Elevation in Tanis Expression Alters Glucose Metabolism and Insulin Sensitivity in H4IIE Cells Diabetes April 2003 52:929-934; doi:10.2337/diabetes.52.4.929
Genetic Liability of Type 1 Diabetes and the Onset Age Among 22,650 Young Finnish Twin Pairs: A Nationwide Follow-Up Study Diabetes April 2003 52:1052-1055; doi:10.2337/diabetes.52.4.1052
Evaluation of Autofluorescent Property of Hemoglobin-Advanced Glycation End Product as a Long-Term Glycemic Index of Diabetes
Diabetes April 2003 52:1041-1046; doi:10.2337/diabetes.52.4.1041
Human Metabolic Syndrome Resulting From Dominant-Negative Mutations in the Nuclear Receptor Peroxisome Proliferator-Activated
Receptor-γ Diabetes April 2003 52:910-917; doi:10.2337/diabetes.52.4.910
Protein Kinase C δ Activation and Translocation to the Nucleus Are Required for Fatty Acid-Induced Apoptosis of Insulin-Secreting
Cells Diabetes April 2003 52:991-997; doi:10.2337/diabetes.52.4.991
Ghrelin Directly Interacts With Neuropeptide-Y-Containing Neurons in the Rat Arcuate Nucleus: Ca2+ Signaling via Protein Kinase A and N-Type Channel-Dependent Mechanisms and Cross-Talk With Leptin and Orexin
Diabetes April 2003 52:948-956; doi:10.2337/diabetes.52.4.948
Genetic Liability of Type 1 Diabetes and the Onset Age Among 22,650 Young Finnish Twin Pairs: A Nationwide Follow-Up Study Diabetes April 2003 52:1052-1055; doi:10.2337/diabetes.52.4.1052
Ghrelin Directly Interacts With Neuropeptide-Y-Containing Neurons in the Rat Arcuate Nucleus: Ca2+ Signaling via Protein Kinase A and N-Type Channel-Dependent Mechanisms and Cross-Talk With Leptin and Orexin
Diabetes April 2003 52:948-956; doi:10.2337/diabetes.52.4.948
Inhibition of Mitochondrial Na+-Ca2+ Exchanger Increases Mitochondrial Metabolism and Potentiates Glucose-Stimulated Insulin Secretion in Rat Pancreatic Islets
Diabetes April 2003 52:965-973; doi:10.2337/diabetes.52.4.965
Genetic Liability of Type 1 Diabetes and the Onset Age Among 22,650 Young Finnish Twin Pairs: A Nationwide Follow-Up Study Diabetes April 2003 52:1052-1055; doi:10.2337/diabetes.52.4.1052
Inhibition of Mitochondrial Na+-Ca2+ Exchanger Increases Mitochondrial Metabolism and Potentiates Glucose-Stimulated Insulin Secretion in Rat Pancreatic Islets
Diabetes April 2003 52:965-973; doi:10.2337/diabetes.52.4.965
Inhibition of Mitochondrial Na+-Ca2+ Exchanger Increases Mitochondrial Metabolism and Potentiates Glucose-Stimulated Insulin Secretion in Rat Pancreatic Islets
Diabetes April 2003 52:965-973; doi:10.2337/diabetes.52.4.965
Differential Activation Mechanisms of Erk-1/2 and p70S6K by Glucose in Pancreatic β-Cells
Diabetes April 2003 52:974-983; doi:10.2337/diabetes.52.4.974
Inhibition of Mitochondrial Na+-Ca2+ Exchanger Increases Mitochondrial Metabolism and Potentiates Glucose-Stimulated Insulin Secretion in Rat Pancreatic Islets
Diabetes April 2003 52:965-973; doi:10.2337/diabetes.52.4.965
Nephrin Expression Is Reduced in Human Diabetic Nephropathy: Evidence for a Distinct Role for Glycated Albumin and Angiotensin II Diabetes April 2003 52:1023-1030; doi:10.2337/diabetes.52.4.1023
Inhibition of Mitochondrial Na+-Ca2+ Exchanger Increases Mitochondrial Metabolism and Potentiates Glucose-Stimulated Insulin Secretion in Rat Pancreatic Islets
Diabetes April 2003 52:965-973; doi:10.2337/diabetes.52.4.965
Mechanisms for the Deterioration in Glucose Tolerance Associated With HIV Protease Inhibitor Regimens Diabetes April 2003 52:918-925; doi:10.2337/diabetes.52.4.918
Is Podocyte Injury Relevant in Diabetic Nephropathy?: Studies in Patients With Type 2 Diabetes Diabetes April 2003 52:1031-1035; doi:10.2337/diabetes.52.4.1031
Low Glomerular Filtration Rate in Normoalbuminuric Type 1 Diabetic Patients: An Indicator of More Advanced Glomerular Lesions Diabetes April 2003 52:1036-1040; doi:10.2337/diabetes.52.4.1036
Mechanisms for the Deterioration in Glucose Tolerance Associated With HIV Protease Inhibitor Regimens Diabetes April 2003 52:918-925; doi:10.2337/diabetes.52.4.918
Inhibition of Mitochondrial Na+-Ca2+ Exchanger Increases Mitochondrial Metabolism and Potentiates Glucose-Stimulated Insulin Secretion in Rat Pancreatic Islets
Diabetes April 2003 52:965-973; doi:10.2337/diabetes.52.4.965
Protein Kinase C δ Activation and Translocation to the Nucleus Are Required for Fatty Acid-Induced Apoptosis of Insulin-Secreting
Cells Diabetes April 2003 52:991-997; doi:10.2337/diabetes.52.4.991
Evaluation of Autofluorescent Property of Hemoglobin-Advanced Glycation End Product as a Long-Term Glycemic Index of Diabetes
Diabetes April 2003 52:1041-1046; doi:10.2337/diabetes.52.4.1041
Ghrelin Directly Interacts With Neuropeptide-Y-Containing Neurons in the Rat Arcuate Nucleus: Ca2+ Signaling via Protein Kinase A and N-Type Channel-Dependent Mechanisms and Cross-Talk With Leptin and Orexin
Diabetes April 2003 52:948-956; doi:10.2337/diabetes.52.4.948
Evaluation of Autofluorescent Property of Hemoglobin-Advanced Glycation End Product as a Long-Term Glycemic Index of Diabetes
Diabetes April 2003 52:1041-1046; doi:10.2337/diabetes.52.4.1041
Inhibition of Mitochondrial Na+-Ca2+ Exchanger Increases Mitochondrial Metabolism and Potentiates Glucose-Stimulated Insulin Secretion in Rat Pancreatic Islets
Diabetes April 2003 52:965-973; doi:10.2337/diabetes.52.4.965
Human Metabolic Syndrome Resulting From Dominant-Negative Mutations in the Nuclear Receptor Peroxisome Proliferator-Activated
Receptor-γ Diabetes April 2003 52:910-917; doi:10.2337/diabetes.52.4.910
Mechanisms for the Deterioration in Glucose Tolerance Associated With HIV Protease Inhibitor Regimens Diabetes April 2003 52:918-925; doi:10.2337/diabetes.52.4.918
Elevation in Tanis Expression Alters Glucose Metabolism and Insulin Sensitivity in H4IIE Cells Diabetes April 2003 52:929-934; doi:10.2337/diabetes.52.4.929
Mechanisms for the Deterioration in Glucose Tolerance Associated With HIV Protease Inhibitor Regimens Diabetes April 2003 52:918-925; doi:10.2337/diabetes.52.4.918
Differential Activation Mechanisms of Erk-1/2 and p70S6K by Glucose in Pancreatic β-Cells
Diabetes April 2003 52:974-983; doi:10.2337/diabetes.52.4.974
Protein Kinase C δ Activation and Translocation to the Nucleus Are Required for Fatty Acid-Induced Apoptosis of Insulin-Secreting
Cells Diabetes April 2003 52:991-997; doi:10.2337/diabetes.52.4.991
Defective Signaling Through Akt-2 and -3 But Not Akt-1 in Insulin-Resistant Human Skeletal Muscle: Potential Role in Insulin Resistance Diabetes April 2003 52:935-941; doi:10.2337/diabetes.52.4.935
Is Podocyte Injury Relevant in Diabetic Nephropathy?: Studies in Patients With Type 2 Diabetes Diabetes April 2003 52:1031-1035; doi:10.2337/diabetes.52.4.1031
Nephrin Expression Is Reduced in Human Diabetic Nephropathy: Evidence for a Distinct Role for Glycated Albumin and Angiotensin II Diabetes April 2003 52:1023-1030; doi:10.2337/diabetes.52.4.1023
Is Podocyte Injury Relevant in Diabetic Nephropathy?: Studies in Patients With Type 2 Diabetes Diabetes April 2003 52:1031-1035; doi:10.2337/diabetes.52.4.1031
Nephrin Expression Is Reduced in Human Diabetic Nephropathy: Evidence for a Distinct Role for Glycated Albumin and Angiotensin II Diabetes April 2003 52:1023-1030; doi:10.2337/diabetes.52.4.1023
Human Metabolic Syndrome Resulting From Dominant-Negative Mutations in the Nuclear Receptor Peroxisome Proliferator-Activated
Receptor-γ Diabetes April 2003 52:910-917; doi:10.2337/diabetes.52.4.910
Protein Kinase C δ Activation and Translocation to the Nucleus Are Required for Fatty Acid-Induced Apoptosis of Insulin-Secreting
Cells Diabetes April 2003 52:991-997; doi:10.2337/diabetes.52.4.991
Elevation in Tanis Expression Alters Glucose Metabolism and Insulin Sensitivity in H4IIE Cells Diabetes April 2003 52:929-934; doi:10.2337/diabetes.52.4.929
Human Metabolic Syndrome Resulting From Dominant-Negative Mutations in the Nuclear Receptor Peroxisome Proliferator-Activated
Receptor-γ Diabetes April 2003 52:910-917; doi:10.2337/diabetes.52.4.910
Elevation in Tanis Expression Alters Glucose Metabolism and Insulin Sensitivity in H4IIE Cells Diabetes April 2003 52:929-934; doi:10.2337/diabetes.52.4.929
Human Metabolic Syndrome Resulting From Dominant-Negative Mutations in the Nuclear Receptor Peroxisome Proliferator-Activated
Receptor-γ Diabetes April 2003 52:910-917; doi:10.2337/diabetes.52.4.910
Genetic Liability of Type 1 Diabetes and the Onset Age Among 22,650 Young Finnish Twin Pairs: A Nationwide Follow-Up Study Diabetes April 2003 52:1052-1055; doi:10.2337/diabetes.52.4.1052
Human Metabolic Syndrome Resulting From Dominant-Negative Mutations in the Nuclear Receptor Peroxisome Proliferator-Activated
Receptor-γ Diabetes April 2003 52:910-917; doi:10.2337/diabetes.52.4.910
Inhibition of Mitochondrial Na+-Ca2+ Exchanger Increases Mitochondrial Metabolism and Potentiates Glucose-Stimulated Insulin Secretion in Rat Pancreatic Islets
Diabetes April 2003 52:965-973; doi:10.2337/diabetes.52.4.965
Nephrin Expression Is Reduced in Human Diabetic Nephropathy: Evidence for a Distinct Role for Glycated Albumin and Angiotensin II Diabetes April 2003 52:1023-1030; doi:10.2337/diabetes.52.4.1023
Human Metabolic Syndrome Resulting From Dominant-Negative Mutations in the Nuclear Receptor Peroxisome Proliferator-Activated
Receptor-γ Diabetes April 2003 52:910-917; doi:10.2337/diabetes.52.4.910
Elevation in Tanis Expression Alters Glucose Metabolism and Insulin Sensitivity in H4IIE Cells Diabetes April 2003 52:929-934; doi:10.2337/diabetes.52.4.929
Mechanisms for the Deterioration in Glucose Tolerance Associated With HIV Protease Inhibitor Regimens Diabetes April 2003 52:918-925; doi:10.2337/diabetes.52.4.918
Human Metabolic Syndrome Resulting From Dominant-Negative Mutations in the Nuclear Receptor Peroxisome Proliferator-Activated
Receptor-γ Diabetes April 2003 52:910-917; doi:10.2337/diabetes.52.4.910
Mechanisms for the Deterioration in Glucose Tolerance Associated With HIV Protease Inhibitor Regimens Diabetes April 2003 52:918-925; doi:10.2337/diabetes.52.4.918
Inhibition of Mitochondrial Na+-Ca2+ Exchanger Increases Mitochondrial Metabolism and Potentiates Glucose-Stimulated Insulin Secretion in Rat Pancreatic Islets
Diabetes April 2003 52:965-973; doi:10.2337/diabetes.52.4.965
Differential Activation Mechanisms of Erk-1/2 and p70S6K by Glucose in Pancreatic β-Cells
Diabetes April 2003 52:974-983; doi:10.2337/diabetes.52.4.974
Ghrelin Directly Interacts With Neuropeptide-Y-Containing Neurons in the Rat Arcuate Nucleus: Ca2+ Signaling via Protein Kinase A and N-Type Channel-Dependent Mechanisms and Cross-Talk With Leptin and Orexin
Diabetes April 2003 52:948-956; doi:10.2337/diabetes.52.4.948