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Complications

Mechanisms of Amelioration of Glucose-Induced Endothelial Dysfunction Following Inhibition of Protein Kinase C In Vivo

  1. Gregory Booth,
  2. Timothy J. Stalker,
  3. Allan M. Lefer and
  4. Rosario Scalia
  1. From the Department of Physiology, Jefferson Medical College, Thomas Jefferson University, Philadelphia, Pennsylvania
    Diabetes 2002 May; 51(5): 1556-1564. https://doi.org/10.2337/diabetes.51.5.1556
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    Article Information

    vol. 51 no. 5 1556-1564
    DOI 
    https://doi.org/10.2337/diabetes.51.5.1556
    PubMed 
    11978656

    Published By 
    American Diabetes Association
    Print ISSN 
    0012-1797
    Online ISSN 
    1939-327X
    History 
    • Published in print May 1, 2002.

    Copyright & Usage 
    DIABETES

    Author Information

    1. Gregory Booth,
    2. Timothy J. Stalker,
    3. Allan M. Lefer and
    4. Rosario Scalia
    1. From the Department of Physiology, Jefferson Medical College, Thomas Jefferson University, Philadelphia, Pennsylvania
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      Mechanisms of Amelioration of Glucose-Induced Endothelial Dysfunction Following Inhibition of Protein Kinase C In Vivo
      Gregory Booth, Timothy J. Stalker, Allan M. Lefer, Rosario Scalia
      Diabetes May 2002, 51 (5) 1556-1564; DOI: 10.2337/diabetes.51.5.1556

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      Mechanisms of Amelioration of Glucose-Induced Endothelial Dysfunction Following Inhibition of Protein Kinase C In Vivo
      Gregory Booth, Timothy J. Stalker, Allan M. Lefer, Rosario Scalia
      Diabetes May 2002, 51 (5) 1556-1564; DOI: 10.2337/diabetes.51.5.1556
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