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Islet Studies

Development and Characterization of a Glucagon-Like Peptide 1-Albumin Conjugate

The Ability to Activate the Glucagon-Like Peptide 1 Receptor In Vivo

  1. Jung-Guk Kim1,
  2. Laurie L. Baggio1,
  3. Dominique P. Bridon2,
  4. Jean-Paul Castaigne2,
  5. Martin F. Robitaille2,
  6. Lucie Jetté2,
  7. Corinne Benquet2 and
  8. Daniel J. Drucker1
  1. 1Banting and Best Diabetes Centre, Department of Medicine, University of Toronto, Toronto General Hospital, Toronto, Ontario
  2. 2Conjuchem, Montreal, Quebec
    Diabetes 2003 Mar; 52(3): 751-759. https://doi.org/10.2337/diabetes.52.3.751
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    Article Information

    vol. 52 no. 3 751-759
    DOI 
    https://doi.org/10.2337/diabetes.52.3.751
    PubMed 
    12606517

    Published By 
    American Diabetes Association
    Print ISSN 
    0012-1797
    Online ISSN 
    1939-327X
    History 
    • Published in print March 1, 2003.

    Copyright & Usage 
    DIABETES

    Author Information

    1. Jung-Guk Kim1,
    2. Laurie L. Baggio1,
    3. Dominique P. Bridon2,
    4. Jean-Paul Castaigne2,
    5. Martin F. Robitaille2,
    6. Lucie Jetté2,
    7. Corinne Benquet2 and
    8. Daniel J. Drucker1
    1. 1Banting and Best Diabetes Centre, Department of Medicine, University of Toronto, Toronto General Hospital, Toronto, Ontario
    2. 2Conjuchem, Montreal, Quebec
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      Development and Characterization of a Glucagon-Like Peptide 1-Albumin Conjugate
      Jung-Guk Kim, Laurie L. Baggio, Dominique P. Bridon, Jean-Paul Castaigne, Martin F. Robitaille, Lucie Jetté, Corinne Benquet, Daniel J. Drucker
      Diabetes Mar 2003, 52 (3) 751-759; DOI: 10.2337/diabetes.52.3.751

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      Development and Characterization of a Glucagon-Like Peptide 1-Albumin Conjugate
      Jung-Guk Kim, Laurie L. Baggio, Dominique P. Bridon, Jean-Paul Castaigne, Martin F. Robitaille, Lucie Jetté, Corinne Benquet, Daniel J. Drucker
      Diabetes Mar 2003, 52 (3) 751-759; DOI: 10.2337/diabetes.52.3.751
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      • Acyl-Ghrelin Influences Pancreatic β-Cell Function by Interference with KATP Channels
      • Pancreatic β-Cell–Specific Deletion of VPS41 Causes Diabetes Due to Defects in Insulin Secretion
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