Attenuation of Extracellular Matrix Accumulation in Diabetic Nephropathy by the Advanced Glycation End Product Cross-Link Breaker ALT-711 via a Protein Kinase C-α−Dependent Pathway
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vol. 53 no. 11 2921-2930
PubMed
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- August 9, 2004
- September 24, 2003
- Published in print October 25, 2004.
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DIABETES
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- Vicki Thallas-Bonke12,
- Carsten Lindschau3,
- Bishoy Rizkalla1,
- Leon A. Bach2,
- Geoffrey Boner14,
- Matthias Meier3,
- Hermann Haller3,
- Mark E. Cooper12 and
- Josephine M. Forbes12
- 1Danielle Alberti Memorial Centre for Diabetes Complications, Vascular Division, Wynn Domain, Baker Medical Research Institute, Melbourne, Australia
- 2Department of Medicine, University of Melbourne, Austin Hospital, Heidelberg, Australia
- 3Department of Nephrology, Hannover Medical School, Hannover, Germany
- 4Department of Medicine, Sackler Faculty of Medicine, Tel Aviv University, Tel Aviv, Israel
- Address correspondence and reprint requests to Vicki Thallas-Bonke, Diabetes Complications, Vascular Division, Baker Medical Research Institute, P.O. Box 6492, St. Kilda Rd. Central, Melbourne, Australia. E-mail: vicki.thallas{at}baker.edu.au
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November 2004, 53(11)
Attenuation of Extracellular Matrix Accumulation in Diabetic Nephropathy by the Advanced Glycation End Product Cross-Link Breaker ALT-711 via a Protein Kinase C-α−Dependent Pathway
