Upregulation of the Mammalian Target of Rapamycin Complex 1 Pathway by Ras Homolog Enriched in Brain in Pancreatic β-Cells Leads to Increased β-Cell Mass and Prevention of Hyperglycemia
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vol. 58 no. 6 1321-1332
PubMed
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History
- April 18, 2008
- February 25, 2009
- Published in print May 21, 2009.
- Published online ahead of print March 3, 2009.
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- Previous version (March 3, 2009 - 07:30).
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© 2009 by the American Diabetes Association.
Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and the work is not altered. See http://creativecommons.org/licenses/by-nc-nd/3.0/ for details.
Author Information
- Suirin Hamada1,
- Kenta Hara1,
- Takeshi Hamada1,
- Hisafumi Yasuda1,
- Hiroaki Moriyama1,
- Rika Nakayama2,
- Masao Nagata1 and
- Koichi Yokono1
- 1Department of Internal and Geriatric Medicine, Kobe University Graduate School of Medicine, Kobe, Japan;
- 2Laboratory for Animal Resources and Genetic Engineering, Center for Developmental Biology (CDB), RIKEN, Kobe, Japan.
- Corresponding author: Kenta Hara, harak{at}kobe-u.ac.jp.
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June 2009, 58(6)
Upregulation of the Mammalian Target of Rapamycin Complex 1 Pathway by Ras Homolog Enriched in Brain in Pancreatic β-Cells Leads to Increased β-Cell Mass and Prevention of Hyperglycemia
