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Original Article

Short-Term Exercise Training Does Not Stimulate Skeletal Muscle ATP Synthesis in Relatives of Humans With Type 2 Diabetes

  1. Gertrud Kacerovsky-Bielesz1,2,
  2. Marek Chmelik2,3,
  3. Charlotte Ling4,
  4. Rochus Pokan5,
  5. Julia Szendroedi1,2,
  6. Michaela Farukuoye2,
  7. Michaela Kacerovsky2,
  8. Albrecht I. Schmid2,3,
  9. Stephan Gruber3,
  10. Michael Wolzt6,
  11. Ewald Moser3,
  12. Giovanni Pacini7,
  13. Gerhard Smekal5,
  14. Leif Groop4 and
  15. Michael Roden1,2,8
  1. 1Medical Department, Hanusch Hospital, Vienna, Austria;
  2. 2Karl-Landsteiner Institute for Endocrinology and Metabolism, Vienna, Austria;
  3. 3MR Center of Excellence, Medical University of Vienna, Vienna, Austria;
  4. 4Department of Clinical Sciences, Lund University, Malmö, Sweden;
  5. 5Department of Sports and Exercise Physiology, University of Vienna, Vienna, Austria;
  6. 6Department of Clinical Pharmacology, Medical University of Vienna, Vienna, Austria;
  7. 7Metabolic Unit, Institute of Biomedical Engineering, Consiglio Nazionale delle Ricerche, Padua, Italy;
  8. 8Institute for Clinical Diabetology, German Diabetes Center-Leibniz Center for Diabetes Research, Department of Medicine/Metabolic Diseases, Heinrich Heine University Düsseldorf, Düsseldorf, Germany.
  1. Corresponding author: Michael Roden, michael.roden{at}ddz.uni-duesseldorf.de.
Diabetes 2009 Jun; 58(6): 1333-1341. https://doi.org/10.2337/db08-1240
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Article Information

vol. 58 no. 6 1333-1341
DOI 
https://doi.org/10.2337/db08-1240
PubMed 
19265027

Published By 
American Diabetes Association
Print ISSN 
0012-1797
Online ISSN 
1939-327X
History 
  • September 6, 2008
  • February 27, 2009
  • Published in print May 21, 2009.
  • Published online ahead of print March 5, 2009.

Article Versions

  • Previous version (March 5, 2009 - 11:59).
  • You are viewing the most recent version of this article.
Copyright & Usage 
© 2009 by the American Diabetes Association. Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and the work is not altered. See http://creativecommons.org/licenses/by-nc-nd/3.0/ for details.

Author Information

  1. Gertrud Kacerovsky-Bielesz1,2,
  2. Marek Chmelik2,3,
  3. Charlotte Ling4,
  4. Rochus Pokan5,
  5. Julia Szendroedi1,2,
  6. Michaela Farukuoye2,
  7. Michaela Kacerovsky2,
  8. Albrecht I. Schmid2,3,
  9. Stephan Gruber3,
  10. Michael Wolzt6,
  11. Ewald Moser3,
  12. Giovanni Pacini7,
  13. Gerhard Smekal5,
  14. Leif Groop4 and
  15. Michael Roden1,2,8
  1. 1Medical Department, Hanusch Hospital, Vienna, Austria;
  2. 2Karl-Landsteiner Institute for Endocrinology and Metabolism, Vienna, Austria;
  3. 3MR Center of Excellence, Medical University of Vienna, Vienna, Austria;
  4. 4Department of Clinical Sciences, Lund University, Malmö, Sweden;
  5. 5Department of Sports and Exercise Physiology, University of Vienna, Vienna, Austria;
  6. 6Department of Clinical Pharmacology, Medical University of Vienna, Vienna, Austria;
  7. 7Metabolic Unit, Institute of Biomedical Engineering, Consiglio Nazionale delle Ricerche, Padua, Italy;
  8. 8Institute for Clinical Diabetology, German Diabetes Center-Leibniz Center for Diabetes Research, Department of Medicine/Metabolic Diseases, Heinrich Heine University Düsseldorf, Düsseldorf, Germany.
  1. Corresponding author: Michael Roden, michael.roden{at}ddz.uni-duesseldorf.de.
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Short-Term Exercise Training Does Not Stimulate Skeletal Muscle ATP Synthesis in Relatives of Humans With Type 2 Diabetes
Gertrud Kacerovsky-Bielesz, Marek Chmelik, Charlotte Ling, Rochus Pokan, Julia Szendroedi, Michaela Farukuoye, Michaela Kacerovsky, Albrecht I. Schmid, Stephan Gruber, Michael Wolzt, Ewald Moser, Giovanni Pacini, Gerhard Smekal, Leif Groop, Michael Roden
Diabetes Jun 2009, 58 (6) 1333-1341; DOI: 10.2337/db08-1240

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Short-Term Exercise Training Does Not Stimulate Skeletal Muscle ATP Synthesis in Relatives of Humans With Type 2 Diabetes
Gertrud Kacerovsky-Bielesz, Marek Chmelik, Charlotte Ling, Rochus Pokan, Julia Szendroedi, Michaela Farukuoye, Michaela Kacerovsky, Albrecht I. Schmid, Stephan Gruber, Michael Wolzt, Ewald Moser, Giovanni Pacini, Gerhard Smekal, Leif Groop, Michael Roden
Diabetes Jun 2009, 58 (6) 1333-1341; DOI: 10.2337/db08-1240
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Original Article

  • An Update on the Molecular Actions of Fenofibrate and Its Clinical Effects on Diabetic Retinopathy and Other Microvascular End Points in Patients With Diabetes
  • Loss of Prohibitin Induces Mitochondrial Damages Altering β-Cell Function and Survival and Is Responsible for Gradual Diabetes Development
  • Cbl-b Is a Critical Regulator of Macrophage Activation Associated With Obesity-Induced Insulin Resistance in Mice
Show more Original Article

Pathophysiology

  • An Update on the Molecular Actions of Fenofibrate and Its Clinical Effects on Diabetic Retinopathy and Other Microvascular End Points in Patients With Diabetes
  • Loss of Prohibitin Induces Mitochondrial Damages Altering β-Cell Function and Survival and Is Responsible for Gradual Diabetes Development
  • Cbl-b Is a Critical Regulator of Macrophage Activation Associated With Obesity-Induced Insulin Resistance in Mice
Show more Pathophysiology

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