Defects in IL-2R Signaling Contribute to Diminished Maintenance of FOXP3 Expression in CD4+CD25+ Regulatory T-Cells of Type 1 Diabetic Subjects
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vol. 59 no. 2 407-415
PubMed
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History
- Received May 8, 2009
- Accepted October 16, 2009
- Published in print January 26, 2010.
- Published online ahead of print October 29, 2009.
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- Previous version (October 29, 2009 - 12:12).
- You are viewing the most recent version of this article.
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© 2010 by the American Diabetes Association.
Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and the work is not altered. See http://creativecommons.org/licenses/by-nc-nd/3.0/ for details.
Author Information
- S. Alice Long1,
- Karen Cerosaletti1,
- Paul L. Bollyky1,
- Megan Tatum1,
- Heather Shilling1,
- Sheng Zhang2,
- Zhong-Yin Zhang2,
- Catherine Pihoker3,
- Srinath Sanda1,
- Carla Greenbaum1 and
- Jane H. Buckner1
- 1Benaroya Research Institute at Virginia Mason, Seattle, Washington;
- 2Department of Biochemistry and Molecular Biology, Indiana University School of Medicine, Indianapolis, Indiana;
- 3Seattle Children's Hospital, Seattle, Washington.
- Corresponding author: Jane H. Buckner, jbuckner{at}benaroyaresearch.org.
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February 2010, 59(2)
Defects in IL-2R Signaling Contribute to Diminished Maintenance of FOXP3 Expression in CD4+CD25+ Regulatory T-Cells of Type 1 Diabetic Subjects
