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Pharmacology and Therapeutics

Resveratrol Inhibits Neuronal Apoptosis and Elevated Ca2+/Calmodulin-Dependent Protein Kinase II Activity in Diabetic Mouse Retina

  1. Young-Hee Kim,
  2. Yoon-Sook Kim,
  3. Sang-Soo Kang,
  4. Gyeong-Jae Cho and
  5. Wan-Sung Choi
  1. From the Department of Anatomy and Neurobiology, Gyeongsang National University, Jinju, Gyeongnam, Korea.
  1. Corresponding author: Wan Sung Choi, choiws{at}gnu.ac.kr.
Diabetes 2010 Jul; 59(7): 1825-1835. https://doi.org/10.2337/db09-1431
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Article Information

vol. 59 no. 7 1825-1835
DOI 
https://doi.org/10.2337/db09-1431
PubMed 
20424226

Published By 
American Diabetes Association
Print ISSN 
0012-1797
Online ISSN 
1939-327X
History 
  • Received September 25, 2009
  • Accepted April 17, 2010
  • Published in print June 29, 2010.
  • Published online ahead of print April 27, 2010.

Article Versions

  • Previous version (April 27, 2010 - 12:47).
  • You are viewing the most recent version of this article.
Copyright & Usage 
© 2010 by the American Diabetes Association. Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and the work is not altered. See http://creativecommons.org/licenses/by-nc-nd/3.0/ for details.

Author Information

  1. Young-Hee Kim,
  2. Yoon-Sook Kim,
  3. Sang-Soo Kang,
  4. Gyeong-Jae Cho and
  5. Wan-Sung Choi
  1. From the Department of Anatomy and Neurobiology, Gyeongsang National University, Jinju, Gyeongnam, Korea.
  1. Corresponding author: Wan Sung Choi, choiws{at}gnu.ac.kr.
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Resveratrol Inhibits Neuronal Apoptosis and Elevated Ca2+/Calmodulin-Dependent Protein Kinase II Activity in Diabetic Mouse Retina
Young-Hee Kim, Yoon-Sook Kim, Sang-Soo Kang, Gyeong-Jae Cho, Wan-Sung Choi
Diabetes Jul 2010, 59 (7) 1825-1835; DOI: 10.2337/db09-1431

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Resveratrol Inhibits Neuronal Apoptosis and Elevated Ca2+/Calmodulin-Dependent Protein Kinase II Activity in Diabetic Mouse Retina
Young-Hee Kim, Yoon-Sook Kim, Sang-Soo Kang, Gyeong-Jae Cho, Wan-Sung Choi
Diabetes Jul 2010, 59 (7) 1825-1835; DOI: 10.2337/db09-1431
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