Ask1 Gene Deletion Blocks Maternal Diabetes–Induced Endoplasmic Reticulum Stress in the Developing Embryo by Disrupting the Unfolded Protein Response Signalosome
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vol. 64 no. 3 973-988
PubMed
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History
- Received March 12, 2014
- Accepted September 16, 2014
- Published in print February 24, 2015.
- Published online ahead of print September 23, 2014.
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- Previous version (September 23, 2014 - 06:10).
- You are viewing the most recent version of this article.
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© 2015 by the American Diabetes Association. Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and the work is not altered.
Author Information
- Fang Wang1,
- Yanqing Wu1,
- Hui Gu1,
- E. Albert Reece1,2,
- Shengyun Fang2,3,4,
- Rinat Gabbay-Benziv1,
- Graham Aberdeen1 and
- Peixin Yang1,2⇑
- 1Department of Obstetrics, Gynecology and Reproductive Sciences, University of Maryland School of Medicine, Baltimore, MD
- 2Department of Biochemistry and Molecular Biology, University of Maryland School of Medicine, Baltimore, MD
- 3Center for Biomedical Engineering and Technology, University of Maryland School of Medicine, Baltimore, MD
- 4Department of Physiology, University of Maryland School of Medicine, Baltimore, MD
- Corresponding author: Peixin Yang, pyang{at}fpi.umaryland.edu.
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March 2015, 64(3)
Ask1 Gene Deletion Blocks Maternal Diabetes–Induced Endoplasmic Reticulum Stress in the Developing Embryo by Disrupting the Unfolded Protein Response Signalosome
