Deletion of Both Rab-GTPase–Activating Proteins TBC14KO and TBC1D4 in Mice Eliminates Insulin- and AICAR-Stimulated Glucose Transport. Diabetes 2015;64:746–759
- Alexandra Chadt,
- Anja Immisch,
- Christian de Wendt,
- Christian Springer,
- Zhou Zhou,
- Torben Stermann,
- Geoffrey D. Holman,
- Dominique Loffing-Cueni,
- Johannes Loffing,
- Hans-Georg Joost and
- Hadi Al-Hasani
During production, two errors were introduced to the article listed above. The first is in the title, which should read “Deletion of Both Rab-GTPase–Activating Proteins TBC1D1 and TBC1D4 in Mice Eliminates Insulin- and AICAR-Stimulated Glucose Transport.” The second is in the first sentence of the abstract, which should read “The Rab-GTPase–activating proteins TBC1D1 and TBC1D4 (AS160) were previously shown to regulate GLUT4 translocation in response to activation of AKT and AMP-dependent kinase.” The editors regret the errors.
The online version reflects these changes.
- © 2015 by the American Diabetes Association. Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and the work is not altered.
In this Issue
April 2015, 64(4)
Deletion of Both Rab-GTPase–Activating Proteins TBC14KO and TBC1D4 in Mice Eliminates Insulin- and AICAR-Stimulated Glucose Transport. Diabetes 2015;64:746–759
